All Relations between microtubule-associated protein tau and neuroglial cell

Reference Sentence Publish Date Extraction Date Species
L Faff, A Reichenbach, J Albrech. Two modes of stimulation by ammonia of taurine release from cultured rabbit M\\xc3\\xbcller cells. Neurochemistry international vol 31 issue 2 1997 9220463 a previous study revealed that a 10-min ('acute') treatment of cultured m\xc3\xbcller glia with ammonium ions (further referred to as 'ammonia') at 0.5-5 mm concentration stimulated the release of newly loaded taurine (tau) by a camp-dependent, osmoresistant mechanism. 1997-09-02 2022-01-10 Not clear
L Faff, A Reichenbach, J Albrech. Two modes of stimulation by ammonia of taurine release from cultured rabbit M\\xc3\\xbcller cells. Neurochemistry international vol 31 issue 2 1997 9220463 by contrast, a 10-min treatment with 65 mm kcl, which is a potent, camp-independent stimulus of tau release in untreated m\xc3\xbcller glia, produced no further enhancement of tau release in ammonia-, mb-, h7 or oca-10mg-pretreated cells. 1997-09-02 2022-01-10 Not clear
L Faff, A Reichenbach, J Albrech. Ammonia-induced taurine release from cultured rabbit M\\xc3\\xbcller cells is an osmoresistant process mediated by intracellular accumulation of cyclic AMP. Journal of neuroscience research vol 46 issue 2 1997 8915900 a previous study demonstrated the release of newly loaded radiolabelled taurine (tau) from cultured rabbit m\xc3\xbcller glia not only following typical cell volume-increasing treatments with high (65 mm) potassium ions or hypotonic media, but also with ammonium chloride (further referred to as ammonia), in a dose-dependent manner, at doses ranging from physiological (0.25 mm) to those accompanying hyperammonemic coma (5 mm) (faff-michalak et al., glia 10:114-120, 1994). 1997-03-10 2022-01-10 Not clear
C Bergeron, M S Pollanen, L Weyer, S E Black, A E Lan. Unusual clinical presentations of cortical-basal ganglionic degeneration. Annals of neurology vol 40 issue 6 1997 9007094 cortical-basal ganglionic degeneration classically presents predominantly as a motor disorder with a unique constellation of histological alterations characterized by the presence of neuronal loss and gliosis in a selective distribution, swollen achromasic neurons, and extensive deposition of abnormal tau in neurons and glia. 1997-02-13 2022-01-10 Not clear
T Hanihara, N Amano, T Takahashi, H Nagatomo, S Yagashit. Distribution of tangles and threads in the cerebral cortex in progressive supranuclear palsy. Neuropathology and applied neurobiology vol 21 issue 4 1996 7494600 in immunohistochemical studies, argentophilic glia and threads were mostly positive for tau 2, and a small number of them were weakly positive for ubiquitin and paired helical filament protein. 1996-01-11 2022-01-10 Not clear
M Nishimura, H Tomimoto, T Suenaga, Y Namba, K Ikeda, I Akiguchi, J Kimur. Immunocytochemical characterization of glial fibrillary tangles in Alzheimer's disease brain. The American journal of pathology vol 146 issue 5 1995 7747799 the tangles in glial cells were immunolabeled by antibodies against tau and ubiquitin, and double immunocytochemistry analyzed by confocal laser scanning microscopy demonstrated that the cytoplasms of tangle-bearing glia were labeled by antibodies against transferrin and 2'3'-cyclic nucleotide 3'-phosphohydrolase. 1995-06-15 2022-01-10 Not clear
U L\\xc3\\xbcbke, J Six, M Villanova, J Boons, M Vandermeeren, C Ceuterick, P Cras, J J Marti. Microtubule-associated protein tau epitopes are present in fiber lesions in diverse muscle disorders. The American journal of pathology vol 145 issue 1 1994 7518193 although tau is predominantly a neuronal protein, it has been demonstrated in glia and other nonneuronal cells. 1994-08-08 2022-01-10 Not clear
J Lowe, R J Mayer, M Lando. Ubiquitin in neurodegenerative diseases. Brain pathology (Zurich, Switzerland) vol 3 issue 1 1994 8269084 neurofibrillary tangles in diverse diseases are associated with ubiquitin as are several other tau containing inclusions in both neurones and glia. 1994-01-28 2022-01-10 Not clear
H Hayashi, Y Watanabe, T Shibuy. Relation between long-lasting amounts of excitatory amino acid and its neuronal uptake system in cultured cerebellar granule cells under hypoglycemia. Progress in neuro-psychopharmacology & biological psychiatry vol 17 issue 3 1993 8475325 moreover, the amounts of glu in a glia poor culture of granule cells tended to be higher than those in glia rich cultured cells, while gln, gly and tau concentrations were lower in glia poor cells than in glia rich cells. 1993-05-17 2022-01-10 Not clear
H Hayashi, Y Watanabe, T Shibuy. Relation between long-lasting amounts of excitatory amino acid and its neuronal uptake system in cultured cerebellar granule cells under hypoglycemia. Progress in neuro-psychopharmacology & biological psychiatry vol 17 issue 3 1993 8475325 however, the amounts of glu at 10 and 14 div, and also gly and tau amounts at 10 div were significantly higher than those seen in the cultures of glia poor cells under normal conditions. 1993-05-17 2022-01-10 Not clear
L I Binder, A Frankfurter, L I Rebhu. The distribution of tau in the mammalian central nervous system. The Journal of cell biology vol 101 issue 4 1985 3930508 tau localization was not observed in glia, which indicated that, at least in brain, tau is neuron specific. 1985-11-07 2022-01-10 Not clear
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