| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| J C Fowler, L D Partridge, L Gervit. Hydroxylamine blocks adenosine A1 receptor-mediated inhibition of synaptic transmission in rat hippocampus. Brain research. vol 815. issue 2. 1999-02-26. PMID:9878859. |
the nitric oxide donor hydroxylamine (nh2oh) induced a transient depression of the evoked synaptic potential recorded in the rat hippocampal ca1 region. |
1999-02-26 |
2023-08-12 |
rat |
| T Kleppisch, A Pfeifer, P Klatt, P Ruth, A Montkowski, R Fässler, F Hofman. Long-term potentiation in the hippocampal CA1 region of mice lacking cGMP-dependent kinases is normal and susceptible to inhibition of nitric oxide synthase. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 19. issue 1. 1999-02-05. PMID:9870937. |
the retrograde messenger nitric oxide (no) is thought to induce ltp in the ca1 region of the hippocampus via activation of soluble guanylyl cyclase (sgc) and, ultimately, cgmp-dependent protein kinase (cgk). |
1999-02-05 |
2023-08-12 |
mouse |
| M Onitsuka, S Mihara, S Yamamoto, M Shigemori, H Higash. Nitric oxide contributes to irreversible membrane dysfunction caused by experimental ischemia in rat hippocampal CA1 neurons. Neuroscience research. vol 30. issue 1. 1998-06-16. PMID:9572575. |
nitric oxide contributes to irreversible membrane dysfunction caused by experimental ischemia in rat hippocampal ca1 neurons. |
1998-06-16 |
2023-08-12 |
rat |
| Y Z Xu, D Y Ruan, Y Wu, Y B Jiang, S Y Chen, J Chen, P Sh. Nitric oxide affects LTP in area CA1 and CA3 of hippocampus in low-level lead-exposed rat. Neurotoxicology and teratology. vol 20. issue 1. 1998-04-23. PMID:9511171. |
nitric oxide affects ltp in area ca1 and ca3 of hippocampus in low-level lead-exposed rat. |
1998-04-23 |
2023-08-12 |
rat |
| C C Huang, K S Hs. Nitric oxide signalling is required for the generation of anoxia-induced long-term potentiation in the hippocampus. The European journal of neuroscience. vol 9. issue 10. 1998-02-04. PMID:9421180. |
the involvement of nitric oxide in anoxia-induced long-term potentiation (anoxic ltp) of synaptic transmission was investigated in ca1 neurons of rat hippocampal slices using intracellular recording techniques in vitro. |
1998-02-04 |
2023-08-12 |
rat |
| C C Huang, K S Hs. Nitric oxide signalling is required for the generation of anoxia-induced long-term potentiation in the hippocampus. The European journal of neuroscience. vol 9. issue 10. 1998-02-04. PMID:9421180. |
these results suggest that nitric oxide is required for the generation of anoxia-induced ltp of glutamatergic synaptic transmission in the ca1 region of the rat hippocampus. |
1998-02-04 |
2023-08-12 |
rat |
| W Musleh, S Yaghoubi, M Baudr. Effects of a nitric oxide synthase inhibitor on NMDA receptor function in organotypic hippocampal cultures. Brain research. vol 770. issue 1-2. 1998-01-21. PMID:9372233. |
nitric oxide (no) has been proposed to trigger long-term potentiation (ltp) at ca3 to ca1 synapses. |
1998-01-21 |
2023-08-12 |
Not clear |
| T Nakagomi, H Kanemitsu, K Takagi, E Morikawa, T Kirino, A Tamur. Effect of L-arginine and NG-nitro-L-arginine on delayed neuronal death in the gerbil hippocampus. Neurological research. vol 19. issue 4. 1997-09-29. PMID:9263225. |
to assess the role of nitric oxide (no) in cerebral ischemia, we investigated the effect of l-arginine, a substrate of no synthase (nos), and ng-nitro-l-arginine (l-nna), a nos inhibitor, on neuronal death in the ca1 hippocampal region. |
1997-09-29 |
2023-08-12 |
Not clear |
| K Kohno, T Higuchi, S Ohta, K Kohno, Y Kumon, S Sakak. Neuroprotective nitric oxide synthase inhibitor reduces intracellular calcium accumulation following transient global ischemia in the gerbil. Neuroscience letters. vol 224. issue 1. 1997-05-01. PMID:9132680. |
by observing the ultrastructural intracellular ca2+ distribution with ca(2+)-oxalate-pyroantimonate method, we examined whether the protective mechanism of the nitric oxide (no) synthase inhibitor, n omega-nitro-l-arginine (lnna), involves change of the intracellular ca2+ movement in delayed neuronal death (dnd) in gerbil hippocampal ca1 neurons following 5-min forebrain ischemia. |
1997-05-01 |
2023-08-12 |
Not clear |
| T Ohmori, Y Hirashima, M Kurimoto, S Endo, A Takak. In vitro hypoxia of cortical and hippocampal CA1 neurons: glutamate, nitric oxide, and platelet activating factor participate in the mechanism of selective neural death in CA1 neurons. Brain research. vol 743. issue 1-2. 1997-04-17. PMID:9017237. |
in vitro hypoxia of cortical and hippocampal ca1 neurons: glutamate, nitric oxide, and platelet activating factor participate in the mechanism of selective neural death in ca1 neurons. |
1997-04-17 |
2023-08-12 |
rat |
| T Ohmori, Y Hirashima, M Kurimoto, S Endo, A Takak. In vitro hypoxia of cortical and hippocampal CA1 neurons: glutamate, nitric oxide, and platelet activating factor participate in the mechanism of selective neural death in CA1 neurons. Brain research. vol 743. issue 1-2. 1997-04-17. PMID:9017237. |
these results suggest that ca1 neurons are more sensitive to hypoxia than cerebral cortical neurons, and glutamate, nitric oxide, and paf may participate in the mechanism of selective neural death in neurons of the ca1 region due to hypoxia. |
1997-04-17 |
2023-08-12 |
rat |
| K Kohno, S Ohta, K Kohno, Y Kumon, A Mitani, S Sakaki, K Kataok. Nitric oxide synthase inhibitor reduces delayed neuronal death in gerbil hippocampal CA1 neurons after transient global ischemia without reduction of brain temperature or extracellular glutamate concentration. Brain research. vol 738. issue 2. 1997-04-03. PMID:8955523. |
we planned a study to determine whether or not the mechanism of nitric oxide (no) neurotoxicity involves the elevation of extracellular glutamate or changes of brain temperature in the pathogenesis of delayed neuronal death of gerbil hippocampal ca1 neurons following 5-min transient forebrain ischemia. |
1997-04-03 |
2023-08-12 |
Not clear |
| O Arancio, M Kiebler, C J Lee, V Lev-Ram, R Y Tsien, E R Kandel, R D Hawkin. Nitric oxide acts directly in the presynaptic neuron to produce long-term potentiation in cultured hippocampal neurons. Cell. vol 87. issue 6. 1997-01-28. PMID:8978607. |
nitric oxide (no) has been proposed to act as a retrograde messenger during long-term potentiation (ltp) in the ca1 region of hippocampus, but the inaccessibility of the presynaptic terminal has prevented a definitive test of this hypothesis. |
1997-01-28 |
2023-08-12 |
Not clear |
| D B Kantor, M Lanzrein, S J Stary, G M Sandoval, W B Smith, B M Sullivan, N Davidson, E M Schuma. A role for endothelial NO synthase in LTP revealed by adenovirus-mediated inhibition and rescue. Science (New York, N.Y.). vol 274. issue 5293. 1997-01-03. PMID:8939872. |
pharmacological studies support the idea that nitric oxide (no) serves as a retrograde messenger during long-term potentiation (ltp) in area ca1 of the hippocampus. |
1997-01-03 |
2023-08-12 |
mouse |
| Y Izumi, A M Benz, D B Clifford, C F Zorumsk. Nitric oxide inhibitors attenuate ischemic degeneration in the CA1 region of rat hippocampal slices. Neuroscience letters. vol 210. issue 3. 1996-12-05. PMID:8805119. |
nitric oxide inhibitors attenuate ischemic degeneration in the ca1 region of rat hippocampal slices. |
1996-12-05 |
2023-08-12 |
rat |
| C L Boulton, E Southam, J Garthwait. Nitric oxide-dependent long-term potentiation is blocked by a specific inhibitor of soluble guanylyl cyclase. Neuroscience. vol 69. issue 3. 1996-04-18. PMID:8596640. |
for example, long-term potentiation in the ca1 region of the rat hippocampus was reported to be blocked by inhibitors of nitric oxide synthase and exogenous nitric oxide has been claimed to induce an enduring enhancement of synaptic strength under certain conditions. |
1996-04-18 |
2023-08-12 |
rat |
| K Kohno, S Ohta, S Furuta, K Kohno, Y Kumon, S Sakak. Intraventricular administration of nitric oxide synthase inhibitors prevents delayed neuronal death in gerbil hippocampal CA1 neurons. Neuroscience letters. vol 199. issue 1. 1996-03-19. PMID:8584229. |
we performed experiments to investigate the participation of nitric oxide (no) in the delayed neuronal death (dnd) of gerbil hippocampal ca1 neurons, following 5-min forebrain ischemia with pretreatment of stereotaxic intraventricular administration of several types of no synthase inhibitors and biologically inactive control drugs. |
1996-03-19 |
2023-08-12 |
Not clear |
| P J Zhu, G Erdeml. Nitric oxide may be a mediator of effects of prolonged but not brief anoxia in CA1 neurons in slices. Neuropharmacology. vol 34. issue 1. 1995-08-29. PMID:7623965. |
nitric oxide may be a mediator of effects of prolonged but not brief anoxia in ca1 neurons in slices. |
1995-08-29 |
2023-08-12 |
rat |
| S Shibata, Y Yamamoto, T Tanaka, S Watanab. NG-nitro-L-arginine protects against hypoxia/hypoglycemia-induced decrease in CA1 presynaptic spikes in rat hippocampal slices. European journal of pharmacology. vol 273. issue 3. 1995-06-08. PMID:7537683. |
the effects of nitric oxide (no) synthase inhibitors on the hypoxia/hypoglycemia-induced decrease in ca1 presynaptic fiber spikes elicited by stimulation of the schaffer collaterals were investigated using rat hippocampal slices. |
1995-06-08 |
2023-08-12 |
rat |
| M Endoh, K Maiese, J A Wagne. Expression of the neural form of nitric oxide synthase by CA1 hippocampal neurons and other central nervous system neurons. Neuroscience. vol 63. issue 3. 1995-04-26. PMID:7534883. |
in particular, nitric oxide synthase, the enzyme responsible for the synthesis of nitric oxide, is expressed in the ca1 region of the hippocampus, where there is evidence that nitric oxide may play a major role in long-term potentiation. |
1995-04-26 |
2023-08-12 |
Not clear |