| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| L W Chiang, F E Schweizer, R W Tsien, H Schulma. Nitric oxide synthase expression in single hippocampal neurons. Brain research. Molecular brain research. vol 27. issue 1. 1995-04-06. PMID:7533235. |
these results confirm immunocytochemical data that nos is present in ca1, and, therefore, nitric oxide could function as a retrograde messenger in long-term potentiation. |
1995-04-06 |
2023-08-12 |
rat |
| C L Boulton, A J Irving, E Southam, B Potier, J Garthwaite, G L Collingridg. The nitric oxide--cyclic GMP pathway and synaptic depression in rat hippocampal slices. The European journal of neuroscience. vol 6. issue 10. 1995-03-16. PMID:7850017. |
the ability of exogenous nitric oxide (no) to modify synaptic transmission was investigated in area ca1 of the rat hippocampal slice. |
1995-03-16 |
2023-08-12 |
rat |
| G Segovia, A Porras, F Mor. Effects of a nitric oxide donor on glutamate and GABA release in striatum and hippocampus of the conscious rat. Neuroreport. vol 5. issue 15. 1995-03-06. PMID:7531005. |
the effects of intracerebral perfusion of the nitric oxide (no) donor 3-morpholino-sydnonimine (sin-1) and the nitric oxide synthase inhibitor n-nitroarginine (narg) on the extracellular concentrations of glutamate (glu) and gamma-aminobutyric acid (gaba) in striatum and ca1 area of the hippocampus were studied. |
1995-03-06 |
2023-08-12 |
rat |
| E M Schuman, M K Meffert, H Schulman, D V Madiso. An ADP-ribosyltransferase as a potential target for nitric oxide action in hippocampal long-term potentiation. Proceedings of the National Academy of Sciences of the United States of America. vol 91. issue 25. 1995-01-11. PMID:7991564. |
recent studies of long-term potentiation (ltp) in the ca1 region of the hippocampus have demonstrated that nitric oxide (no) may be involved in some forms of ltp and have suggested that postsynaptically generated no is a candidate to act as a retrograde messenger. |
1995-01-11 |
2023-08-12 |
Not clear |
| A M Buchan, S Z Gertler, Z G Huang, H Li, K E Chaundy, D Xu. Failure to prevent selective CA1 neuronal death and reduce cortical infarction following cerebral ischemia with inhibition of nitric oxide synthase. Neuroscience. vol 61. issue 1. 1994-11-28. PMID:7526264. |
we investigated the putative role of nitric oxide in the expression of neuronal injury following both transient severe forebrain ischemia (ca1 neuronal injury) and transient or permanent middle cerebral artery occlusion (neocortical pannecrosis). |
1994-11-28 |
2023-08-12 |
rat |
| R A Wallis, K L Panizzon, D Henry, C G Wasterlai. Neuroprotection against nitric oxide injury with inhibitors of ADP-ribosylation. Neuroreport. vol 5. issue 3. 1994-03-07. PMID:8298082. |
we investigated the effect of nitric oxide (no) upon ca1 neurons of the hippocampal slice. |
1994-03-07 |
2023-08-12 |
Not clear |
| K Kato, C F Zorumsk. Nitric oxide inhibitors facilitate the induction of hippocampal long-term potentiation by modulating NMDA responses. Journal of neurophysiology. vol 70. issue 3. 1993-12-17. PMID:7693884. |
the effects of the competitive nitric oxide (no) synthase inhibitor, l-nitroarginine (l-noarg), on synaptically activated n-methyl-d-aspartate (nmda) currents and the induction of long-term potentiation (ltp) were studied in the ca1 region of rat hippocampal slices. |
1993-12-17 |
2023-08-12 |
rat |
| D M Chetkovich, E Klann, J D Sweat. Nitric oxide synthase-independent long-term potentiation in area CA1 of hippocampus. Neuroreport. vol 4. issue 7. 1993-10-08. PMID:7690266. |
nitric oxide synthase-independent long-term potentiation in area ca1 of hippocampus. |
1993-10-08 |
2023-08-12 |
Not clear |
| J G Valtschanoff, R J Weinberg, V N Kharazia, M Nakane, H H Schmid. Neurons in rat hippocampus that synthesize nitric oxide. The Journal of comparative neurology. vol 331. issue 1. 1993-08-03. PMID:7686569. |
these data suggest that nitric oxide in ca1 may function as a paracrine agent, rather than a spatially precise messenger, in long-term potentiation. |
1993-08-03 |
2023-08-12 |
rat |
| W Y Musleh, K Shahi, M Baudr. Further studies concerning the role of nitric oxide in LTP induction and maintenance. Synapse (New York, N.Y.). vol 13. issue 4. 1993-05-25. PMID:7683145. |
nitric oxide (no) has recently been proposed to act as a retrograde messenger to produce long-term potentiation (ltp) in hippocampal area ca1. |
1993-05-25 |
2023-08-12 |
Not clear |
| R A Wallis, K Panizzon, C G Wasterlai. Inhibition of nitric oxide synthase protects against hypoxic neuronal injury. Neuroreport. vol 3. issue 7. 1992-12-14. PMID:1384768. |
these results indicate that nitric oxide generation may mediate acute ca1 neuronal injury during hypoxia, and that inhibition of nitric oxide synthase may be a useful neuroprotective strategy. |
1992-12-14 |
2023-08-11 |
Not clear |
| Y Izumi, A M Benz, D B Clifford, C F Zorumsk. Nitric oxide inhibitors attenuate N-methyl-D-aspartate excitotoxicity in rat hippocampal slices. Neuroscience letters. vol 135. issue 2. 1992-08-07. PMID:1625799. |
to investigate whether nitric oxide (no) plays a role in the neurotoxicity produced by n-methyl-d-aspartate (nmda) we have examined the effects of no inhibitors on nmda-mediated neurodegeneration in the ca1 region of rat hippocampal slices. |
1992-08-07 |
2023-08-11 |
rat |
| J E Haley, G L Wilcox, P F Chapma. The role of nitric oxide in hippocampal long-term potentiation. Neuron. vol 8. issue 2. 1992-03-26. PMID:1371216. |
we investigated the role of nitric oxide (no) in determining the duration of potentiation induced by high frequency stimulation of afferents in the ca1 region of the rat hippocampus. |
1992-03-26 |
2023-08-11 |
rat |