All Relations between matrix compartment and nitrogen monoxide

Publication Sentence Publish Date Extraction Date Species
M Iovu, G Dumais, P du Souic. Anti-inflammatory activity of chondroitin sulfate. Osteoarthritis and cartilage. vol 16 Suppl 3. 2008-12-04. PMID:18667340. articular damage and synovitis are secondary to local increase of pro-inflammatory cytokines (interleukin-1beta and tumor necrosis factor-alpha), enzymes with proteolytic activity (matrix metalloproteinases), and enzymes with pro-inflammatory activity (cyclooxygenase-2 and nitric oxide synthase-2). 2008-12-04 2023-08-12 Not clear
S Gambaryan, A Kobsar, S Hartmann, I Birschmann, P J Kuhlencordt, W Müller-Esterl, S M Lohmann, U Walte. NO-synthase-/NO-independent regulation of human and murine platelet soluble guanylyl cyclase activity. Journal of thrombosis and haemostasis : JTH. vol 6. issue 8. 2008-12-03. PMID:18485089. platelets, specialized adhesive cells, play key roles in normal and pathological hemostasis through their ability to rapidly adhere to subendothelial matrix proteins (adhesion) and to other activated platelets (aggregation), functions which are inhibited by nitric oxide (no). 2008-12-03 2023-08-12 human
Yi-Ling Yang, Demeral David Liu, Nan-Kuang Hsieh, Hsing I Che. Endothelin and gelatinases in renal changes following blockade of nitric oxide synthase in hypertensive rats. The Chinese journal of physiology. vol 51. issue 3. 2008-11-25. PMID:18935914. we investigated the involvement of matrix metalloproteinases (mmps), tissue inhibitor (timp) and endothelin-1 (et-1) in the renal damage in spontaneously hypertensive rats (shr) following nitric oxide (no) deprivation. 2008-11-25 2023-08-12 rat
A L Stevens, C A Wheeler, S R Tannenbaum, A J Grodzinsk. Nitric oxide enhances aggrecan degradation by aggrecanase in response to TNF-alpha but not IL-1beta treatment at a post-transcriptional level in bovine cartilage explants. Osteoarthritis and cartilage. vol 16. issue 4. 2008-11-03. PMID:17923423. the objective of this study was to determine the role of nitric oxide (no) in tumor necrosis factor alpha (tnf-alpha)-induced matrix damage, compared to interleukin 1 beta (il-1beta), in bovine cartilage explant cultures. 2008-11-03 2023-08-12 cattle
R Tsutsumi, H Ito, T Hiramitsu, K Nishitani, M Akiyoshi, T Kitaori, T Yasuda, T Nakamur. Celecoxib inhibits production of MMP and NO via down-regulation of NF-kappaB and JNK in a PGE2 independent manner in human articular chondrocytes. Rheumatology international. vol 28. issue 8. 2008-10-28. PMID:18080123. the purpose of this study was to examine the effects of celecoxib on matrix metalloproteinases (mmp-1 and mmp-3), nitric oxide (no), and the phosphorylation of nuclear factor-kappab (nf-kappab) and three mitogen-activated protein kinases (mapks), (p38, jnk and erk) in human articular chondrocytes from normal, osteoarthritis, and rheumatoid arthritis cartilages. 2008-10-28 2023-08-12 human
Paavo Korge, Peipei Ping, James N Weis. Reactive oxygen species production in energized cardiac mitochondria during hypoxia/reoxygenation: modulation by nitric oxide. Circulation research. vol 103. issue 8. 2008-10-23. PMID:18776040. consistent with its cardioprotective actions, nitric oxide ( o) abrogated increased h(2)dcf oxidation under these conditions, as well as attenuating ros-induced increases in matrix [fe(2+)] and aconitase inhibition caused by antimycin. 2008-10-23 2023-08-12 Not clear
Hideaki Yamaguchi, Yumi Kidachi, Hironori Umetsu, Kazuo Ryoyam. Hypoxia enhances gene expression of inducible nitric oxide synthase and matrix metalloproteinase-9 in ras/myc-transformed serum-free mouse embryo cells under simulated inflammatory and infectious conditions. Cell biology international. vol 32. issue 8. 2008-10-14. PMID:18474440. ras/myc-transformed serum-free mouse embryo (ras/myc sfme) cells were treated with interferon-gamma (ifn-gamma; 100 u/ml) and/or lipopolysaccharide (lps; 0.5 microg/ml) for 24 h to simulate inflammatory and infectious conditions and investigate their effects on the expression of inducible nitric oxide synthase (inos) mrna, nitric oxide (no) and matrix metalloproteinase-9 (mmp-9). 2008-10-14 2023-08-12 mouse
Camila Oliveira Rodini, Aline Carvalho Batista, Thiago José Dionísio, Carlos Ferreira Santos, Fernando Queiroz Cunha, Vanessa Soares Lar. Morphologic evaluation and expression of matrix metalloproteinases-2 and 9 and nitric oxide during experimental periodontal disease in rat. Journal of molecular histology. vol 39. issue 3. 2008-10-03. PMID:18247145. morphologic evaluation and expression of matrix metalloproteinases-2 and 9 and nitric oxide during experimental periodontal disease in rat. 2008-10-03 2023-08-12 rat
Camila Oliveira Rodini, Aline Carvalho Batista, Thiago José Dionísio, Carlos Ferreira Santos, Fernando Queiroz Cunha, Vanessa Soares Lar. Morphologic evaluation and expression of matrix metalloproteinases-2 and 9 and nitric oxide during experimental periodontal disease in rat. Journal of molecular histology. vol 39. issue 3. 2008-10-03. PMID:18247145. metalloproteinases (mmps) and nitric oxide (no) play essential role in extracellular matrix turnover of periodontal tissue destruction. 2008-10-03 2023-08-12 rat
Michiko Yamada, Yoriko Sankoda, Ryuichi Tatsumi, Wataru Mizunoya, Yoshihide Ikeuchi, Kenji Sunagawa, Ronald E Alle. Matrix metalloproteinase-2 mediates stretch-induced activation of skeletal muscle satellite cells in a nitric oxide-dependent manner. The international journal of biochemistry & cell biology. vol 40. issue 10. 2008-10-02. PMID:18403250. this process depends on nitric oxide (no) production, release of hepatocyte growth factor (hgf) from the extracellular matrix, and presentation of hgf to the c-met receptor. 2008-10-02 2023-08-12 rat
Caroline Demacq, Ingrid F Metzger, Raquel F Gerlach, Jose E Tanus-Santo. Inverse relationship between markers of nitric oxide formation and plasma matrix metalloproteinase-9 levels in healthy volunteers. Clinica chimica acta; international journal of clinical chemistry. vol 394. issue 1-2. 2008-09-05. PMID:18455513. inverse relationship between markers of nitric oxide formation and plasma matrix metalloproteinase-9 levels in healthy volunteers. 2008-09-05 2023-08-12 human
Yingrui Wang-Rosenke, Hans-H Neumayer, Harm Peter. NO signaling through cGMP in renal tissue fibrosis and beyond: key pathway and novel therapeutic target. Current medicinal chemistry. vol 15. issue 14. 2008-08-14. PMID:18537617. nitric oxide (no) produced by endothelial no synthase (nos) in low concentrations is a unique messenger molecule with key homeostatic functions concerning the prevention of pathological vascular and tissue changes such as increases in blood pressure, platelet degranulation, mononuclear cell infiltration, cell proliferation and extracellular matrix protein accumulation. 2008-08-14 2023-08-12 human
Hideaki Yamaguchi, Yumi Kidachi, Hironori Umetsu, Kazuo Ryoyam. L-NAME inhibits tumor cell progression and pulmonary metastasis of r/m HM-SFME-1 cells by decreasing NO from tumor cells and TNF-alpha from macrophages. Molecular and cellular biochemistry. vol 312. issue 1-2. 2008-08-11. PMID:18320293. in addition, production of nitric oxide (no), matrix metalloproteinases (mmps) and tumor necrosis factor-alpha (tnf-alpha) in the tumor cells and in a mouse macrophage-like cell line, j774.1 cells, was analyzed. 2008-08-11 2023-08-12 mouse
Maya S P Huijberts, Nicolaas C Schaper, Casper G Schalkwij. Advanced glycation end products and diabetic foot disease. Diabetes/metabolism research and reviews. vol 24 Suppl 1. 2008-08-07. PMID:18442180. several mechanisms have been proposed by which ages lead to diabetic complications such as the accumulation of ages in the extracellular matrix causing aberrant cross-linking, the binding of circulating ages to the receptor of ages (rage) on different cell types and activation of key cell signalling pathways with subsequent modulation of gene expression, and intracellular age formation leading to quenching of nitric oxide and impaired function of growth factors. 2008-08-07 2023-08-12 Not clear
Hongwei Shao, Yaohong Tan, Darwin Eton, Zhe Yang, M Georgina Uberti, Sen Li, Andrew Schulick, Hong Y. Statin and stromal cell-derived factor-1 additively promote angiogenesis by enhancement of progenitor cells incorporation into new vessels. Stem cells (Dayton, Ohio). vol 26. issue 5. 2008-07-28. PMID:18308946. in vitro mono-treatment with either fluvastatin (100 nm) or sdf-1 (100 ng/ml) facilitated epc proliferation and migration, inhibited epc apoptosis, enhanced expression of matrix metalloproteinase-2 (mmp-2) and -9 (mmp-9), and increased akt phosphorylation and nitric oxide production. 2008-07-28 2023-08-12 mouse
Min-Chul Seo, Sangseop Kim, Sang-Hyun Kim, Long Tai Zheng, Eui Kyun Park, Won-Ha Lee, Kyoungho Su. Discoidin domain receptor 1 mediates collagen-induced inflammatory activation of microglia in culture. Journal of neuroscience research. vol 86. issue 5. 2008-04-23. PMID:17969104. using bv-2 mouse microglial cells and mouse primary microglial cultures, we have demonstrated that (1) collagen induces inflammatory activation of microglia as evidenced by production of nitric oxide, expression of inducible nitric oxide synthase, cox-2, cd40, and matrix metalloproteinase-9; (2) ddr1 is expressed in microglia and is phosphorylated by collagen treatment; and (3) collagen-induced microglial activation is abrogated by ddr1 blockade but not by integrin neutralization. 2008-04-23 2023-08-12 mouse
K M Naseem, R Rib. Unresolved roles of platelet nitric oxide synthase. Journal of thrombosis and haemostasis : JTH. vol 6. issue 1. 2008-02-28. PMID:17944990. endothelial-derived nitric oxide (no) is a key regulator of platelet function, inhibiting both adhesion to the extracellular matrix and aggregation at sites of vascular injury. 2008-02-28 2023-08-12 Not clear
Amy M Skrzypchak, Nathan G Lafayette, Robert H Bartlett, Zhengrong Zhou, Megan C Frost, Mark E Meyerhoff, Melissa M Reynolds, Gail M Annic. Effect of varying nitric oxide release to prevent platelet consumption and preserve platelet function in an in vivo model of extracorporeal circulation. Perfusion. vol 22. issue 3. 2008-02-27. PMID:18018399. nitric oxide released from within a polymer matrix has been proven effective for preventing platelet activation and adhesion onto extracorporeal circuits. 2008-02-27 2023-08-12 rabbit
Hideaki Yamaguchi, Yumi Kidachi, Hironori Umetsu, Kazuo Ryoyam. Ras/myc-transformed serum-free mouse embryo cells under simulated inflammatory and infectious conditions increase levels of nitric oxide and matrix metalloproteinase-9 without a direct association between them. Molecular and cellular biochemistry. vol 306. issue 1-2. 2008-01-29. PMID:17660954. ras/myc-transformed serum-free mouse embryo cells under simulated inflammatory and infectious conditions increase levels of nitric oxide and matrix metalloproteinase-9 without a direct association between them. 2008-01-29 2023-08-12 mouse
Lucian Soane, Sibel Kahraman, Tibor Kristian, Gary Fisku. Mechanisms of impaired mitochondrial energy metabolism in acute and chronic neurodegenerative disorders. Journal of neuroscience research. vol 85. issue 15. 2008-01-22. PMID:17847081. these include direct, reversible inhibition of cytochrome oxidase by nitric oxide, release of mitochondrial cytochrome c, oxidative inhibition of mitochondrial matrix dehydrogenases and adenine nucleotide transport, the availability of nad for dehydrogenase reactions, respiratory uncoupling by activities such as that of the permeability transition pore, and altered mitochondrial structure and intracellular trafficking. 2008-01-22 2023-08-12 Not clear