| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| Stefano Zoccolella, Davide Martino, Giovanni Defazio, Paolo Lamberti, Paolo Livre. Hyperhomocysteinemia in movement disorders: Current evidence and hypotheses. Current vascular pharmacology. vol 4. issue 3. 2006-08-29. PMID:16842141. |
increased hcy plasma levels may have important implications in patients affected by these basal ganglia disturbances, by exerting neurotoxic effects, contributing to neurotransmitter imbalance in motor circuits, and increasing the risk for vascular insults and cognitive dysfunctions. |
2006-08-29 |
2023-08-12 |
Not clear |
| C J Zeis. Neuroanatomical phenotyping in the mouse: the dopaminergic system. Veterinary pathology. vol 42. issue 6. 2006-08-01. PMID:16301571. |
one of these resides in the basal nuclei and their associated projections and utilizes dopamine as a neurotransmitter. |
2006-08-01 |
2023-08-12 |
mouse |
| Cécile Lavoute, Michel Weiss, Jean-Claude Rostai. Effects of repeated hyperbaric nitrogen-oxygen exposures on the striatal dopamine release and on motor disturbances in rats. Brain research. vol 1056. issue 1. 2005-11-21. PMID:16112095. |
repetitive exposures to 1 mpa of nitrogen induced a reversal effect on the dopamine release which suggests a neurochemical change at the level of the neurotransmitter regulation processes of the basal ganglia. |
2005-11-21 |
2023-08-12 |
rat |
| S M Brown, P D Kieffaber, C A Carroll, J L Vohs, J A Tracy, A Shekhar, B F O'Donnell, J E Steinmetz, W P Hetric. Eyeblink conditioning deficits indicate timing and cerebellar abnormalities in schizophrenia. Brain and cognition. vol 58. issue 1. 2005-07-21. PMID:15878730. |
accumulating evidence indicates that individuals with schizophrenia manifest abnormalities in structures (cerebellum and basal ganglia) and neurotransmitter systems (dopamine) linked to internal-timing processes. |
2005-07-21 |
2023-08-12 |
Not clear |
| Giuseppe Ferraro, Pierangelo Sardo, Giuseppe Di Giovanni, Rosaria Fileccia, Vittorio La Grutt. CCK-8S systemic administration blocks the 7-nitroindazole-induced effects on the EEG of striatum and globus pallidus: a FFT analysis in the rat. In vivo (Athens, Greece). vol 18. issue 3. 2005-03-07. PMID:15341187. |
nitric oxide (no) and cholecystokinin (cck) are involved in the modulation of several neurotransmitter systems in the basal ganglia, and a functional interaction between their modulatory effects could be hypothesised. |
2005-03-07 |
2023-08-12 |
rat |
| Takuma Mabuchi, Jacinta Lucero, Anne Feng, James A Koziol, Gregory J del Zopp. Focal cerebral ischemia preferentially affects neurons distant from their neighboring microvessels. Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism. vol 25. issue 2. 2005-03-03. PMID:15678127. |
hence, the [m-n distance] relationship in the normal and ischemic basal ganglia is highly ordered, and distant neurons are consistently perturbed by ischemia, although this is not uniformly dependent on neurotransmitter type. |
2005-03-03 |
2023-08-12 |
human |
| Ann E Kelle. Memory and addiction: shared neural circuitry and molecular mechanisms. Neuron. vol 44. issue 1. 2004-11-23. PMID:15450168. |
these two neurotransmitter systems, widely distributed in many regions of cortex, limbic system, and basal ganglia, appear to play a key integrative role in motivation, learning, and memory, thus modulating adaptive behavior. |
2004-11-23 |
2023-08-12 |
Not clear |
| Chan-Hyung Kim, Min-Seong Koo, Keun-Ah Cheon, Young-Hoon Ryu, Jong-Doo Lee, Hong-Shick Le. Dopamine transporter density of basal ganglia assessed with [123I]IPT SPET in obsessive-compulsive disorder. European journal of nuclear medicine and molecular imaging. vol 30. issue 12. 2004-10-05. PMID:14513291. |
these findings suggest that the dopaminergic neurotransmitter system of the basal ganglia in patients with ocd could be involved in the pathophysiology of ocd. |
2004-10-05 |
2023-08-12 |
Not clear |
| Dennis J Zgaljardic, Andrew Feigi. Neuroimaging of Parkinson's disease and atypical parkinsonism. Current neurology and neuroscience reports. vol 4. issue 4. 2004-09-10. PMID:15217542. |
the basal ganglia and its associated circuitry can be assessed with a variety of neuroimaging methods that can provide information regarding specific neurotransmitter systems, the functional activity of brain regions, and the structural integrity of these regions. |
2004-09-10 |
2023-08-12 |
Not clear |
| Benjamin Gauter, Roman Rukwied, Christoph Konra. Cannabinoid agonists in the treatment of blepharospasm--a case report study. Neuro endocrinology letters. vol 25. issue 1-2. 2004-08-03. PMID:15159681. |
it has been suggested that blepharospasm is neurobiologically based on a dysfunction of the basal ganglia and an impairment of the dopamine neurotransmitter system. |
2004-08-03 |
2023-08-12 |
Not clear |
| M Jenuwein, C Scheller, E Neuen-Jacob, S Sopper, T Tatschner, V ter Meulen, P Riederer, E Koutsilier. Dopamine deficits and regulation of the cAMP second messenger system in brains of simian immunodeficiency virus-infected rhesus monkeys. Journal of neurovirology. vol 10. issue 3. 2004-08-02. PMID:15204921. |
the basal ganglia, structures rich in the neurotransmitter dopamine, are primarily affected during human immunodeficiency virus (hiv) infection. |
2004-08-02 |
2023-08-12 |
human |
| Sonia Darbra, Olivier George, Jean-Jacques Bouyer, Pier-Vincenzo Piazza, Michel Le Moal, Willy May. Sleep-wake states and cortical synchronization control by pregnenolone sulfate into the pedunculopontine nucleus. Journal of neuroscience research. vol 76. issue 5. 2004-07-06. PMID:15139033. |
these neurons are regulated by classical neurotransmitter systems from the pontomesencephalic reticular formation and basal ganglia. |
2004-07-06 |
2023-08-12 |
rat |
| M Glass, A van Dellen, C Blakemore, A J Hannan, R L M Faul. Delayed onset of Huntington's disease in mice in an enriched environment correlates with delayed loss of cannabinoid CB1 receptors. Neuroscience. vol 123. issue 1. 2004-03-23. PMID:14667455. |
we have compared the levels of receptor binding of a range of basal ganglia neurotransmitter receptors believed to be important in hd, in normal mice and r6/1 transgenic hd mice housed in either enriched or standard laboratory environments. |
2004-03-23 |
2023-08-12 |
mouse |
| Daniel J Bonthius, Bahri Karaca. Sydenham's chorea: not gone and not forgotten. Seminars in pediatric neurology. vol 10. issue 1. 2003-11-19. PMID:12785743. |
because the pathogenesis of sydenham's chorea includes the production of anti-basal ganglia antibodies, therapies that modulate immune function or that restore neurotransmitter balance within the basal ganglia may be effective for sydenham's chorea. |
2003-11-19 |
2023-08-12 |
Not clear |
| Jonathan M Brotchi. CB1 cannabinoid receptor signalling in Parkinson's disease. Current opinion in pharmacology. vol 3. issue 1. 2003-07-01. PMID:12550742. |
thus, because activation of basal ganglia cb(1) receptors can modulate neurotransmission and contribute to synaptic plasticity in a manner similar to that described in other brain regions, it also appears that endocannabinoids might modulate cell-cell signalling via effects on neurotransmitter re-uptake and postsynaptic actions mediating cross talk between multiple receptor types. |
2003-07-01 |
2023-08-12 |
Not clear |
| Angelika Richter, Wolfgang Lösche. Effects of pharmacological manipulations of cannabinoid receptors on severity of dystonia in a genetic model of paroxysmal dyskinesia. European journal of pharmacology. vol 454. issue 2-3. 2003-05-06. PMID:12421641. |
in view of previous pathophysiological findings in mutant hamsters, the antidystonic efficacy of win 55,212-2 can be explained by modulation of different neurotransmitter systems within the basal ganglia. |
2003-05-06 |
2023-08-12 |
Not clear |
| Lauren B Krup. Fatigue in multiple sclerosis: definition, pathophysiology and treatment. CNS drugs. vol 17. issue 4. 2003-04-28. PMID:12665396. |
proposed pathological mechanisms of fatigue in ms include neuronal factors such as dysfunction of premotor, limbic, basal ganglia or hypothalamic areas; disruption of the neuroendrocrine axis leading to low arousal; alteration in serotoninergic pathways; changes in neurotransmitter levels; and altered cns functioning caused by a disruption of the immune response. |
2003-04-28 |
2023-08-12 |
Not clear |
| M J Messenger, L G Dawson, S Dut. Changes in metabotropic glutamate receptor 1-8 gene expression in the rodent basal ganglia motor loop following lesion of the nigrostriatal tract. Neuropharmacology. vol 43. issue 2. 2003-02-11. PMID:12213280. |
metabotropic glutamate (mglu) receptors in the basal ganglia motor loop may increase cell excitability (group i) or modulate neurotransmitter release (group i, ii and iii). |
2003-02-11 |
2023-08-12 |
rat |
| J J Canales, C Capper-Loup, D Hu, E S Choe, U Upadhyay, A M Graybie. Shifts in striatal responsivity evoked by chronic stimulation of dopamine and glutamate systems. Brain : a journal of neurology. vol 125. issue Pt 10. 2002-10-29. PMID:12244091. |
to examine functional convergence of dopamine and glutamate neurotransmitter systems in the basal ganglia, we evaluated the long-term effects of chronic stimulation of each of these systems on striatal responses to stimulation of the other. |
2002-10-29 |
2023-08-12 |
rat |
| J J Canales, C Capper-Loup, D Hu, E S Choe, U Upadhyay, A M Graybie. Shifts in striatal responsivity evoked by chronic stimulation of dopamine and glutamate systems. Brain : a journal of neurology. vol 125. issue Pt 10. 2002-10-29. PMID:12244091. |
these findings prompt a model of the basal ganglia function as being regulated by opposing homeostatic dopamine-glutamate neurotransmitter interactions. |
2002-10-29 |
2023-08-12 |
rat |