Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
L E Mello, J Villare. Neuroanatomy of the basal ganglia. The Psychiatric clinics of North America. vol 20. issue 4. 1998-03-26. PMID:9443345. |
this article deals with the neuroanatomic aspects of the basal ganglia with regard to different neurotransmitter systems and to different diseases. |
1998-03-26 |
2023-08-12 |
Not clear |
J L Contreras-Vidal, G E Stelmac. Effects of Parkinsonism on motor control. Life sciences. vol 58. issue 3. 1998-03-12. PMID:9499157. |
it is argued that the variability seen within and between individual patients through the course of this neurological disorder may result from abnormal non-uniform neurotransmitter levels as well as functional segregation of neural populations in the basal ganglia. |
1998-03-12 |
2023-08-12 |
Not clear |
Y Tashiro, J M Drake, S Chakrabortty, T Hattor. Functional injury of cholinergic, GABAergic and dopaminergic systems in the basal ganglia of adult rat with kaolin-induced hydrocephalus. Brain research. vol 770. issue 1-2. 1998-01-21. PMID:9372201. |
the disturbance in balance of these neurotransmitter systems in the basal ganglia may explain some of motor functional disabilities in hydrocephalus. |
1998-01-21 |
2023-08-12 |
rat |
S Galatiot. [Immunohistochemical findings in Huntington's Chorea: report of 9 cases]. Pathologica. vol 88. issue 6. 1997-06-30. PMID:9206776. |
these neuropeptides, in particular menk and sp, are reported to coexist with the inhibitory neurotransmitter gaba in the neurons of basal ganglia. |
1997-06-30 |
2023-08-12 |
Not clear |
J Gibbon, C Malapani, C L Dale, C Galliste. Toward a neurobiology of temporal cognition: advances and challenges. Current opinion in neurobiology. vol 7. issue 2. 1997-06-27. PMID:9142762. |
surgical intervention or diseases of the cerebellum generally result in increased variability in temporal processing, whereas both clock and memory effects are seen for neurotransmitter interventions, lesions and diseases of the basal ganglia. |
1997-06-27 |
2023-08-12 |
Not clear |
J A Kearney, K A Frey, R L Albi. Metabotropic glutamate agonist-induced rotation: a pharmacological, FOS immunohistochemical, and [14C]-2-deoxyglucose autoradiographic study. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 17. issue 11. 1997-06-10. PMID:9151758. |
we sought to determine the mglur subtype(s) involved, the modulation of the rotation by other basal ganglia neurotransmitter systems, and the functional anatomy underlying the rotational behavior. |
1997-06-10 |
2023-08-12 |
rat |
R F Butterworth, L Spahr, S Fontaine, G P Layrargue. Manganese toxicity, dopaminergic dysfunction and hepatic encephalopathy. Metabolic brain disease. vol 10. issue 4. 1996-10-24. PMID:8847990. |
a major cause of pallidal hyperintensity on t1-weighted mri is manganese deposition; chronic manganese intoxication in the absence of liver disease results in pallidal mr signal hyperintensity, in extrapyramidal symptoms and in selective effects on the dopaminergic neurotransmitter system in basal ganglia. |
1996-10-24 |
2023-08-12 |
Not clear |
L Medina, A Reine. Neurotransmitter organization and connectivity of the basal ganglia in vertebrates: implications for the evolution of basal ganglia. Brain, behavior and evolution. vol 46. issue 4-5. 1996-03-01. PMID:8564466. |
neurotransmitter organization and connectivity of the basal ganglia in vertebrates: implications for the evolution of basal ganglia. |
1996-03-01 |
2023-08-12 |
Not clear |
L Medina, A Reine. Neurotransmitter organization and connectivity of the basal ganglia in vertebrates: implications for the evolution of basal ganglia. Brain, behavior and evolution. vol 46. issue 4-5. 1996-03-01. PMID:8564466. |
in addition, we review recent findings on the neurotransmitter organization and connectivity of the basal ganglia in amphibians and fishes, with the goal of assessing whether a basal ganglia showing the same basic features as in amniotes is observed in anamniotes. |
1996-03-01 |
2023-08-12 |
Not clear |
M Herrera-Marschitz, C F Loidl, Z B You, K Andersson, R Silveira, W T O'Connor, M Goin. Neurocircuitry of the basal ganglia studied by monitoring neurotransmitter release. Effects of intracerebral and perinatal asphyctic lesions. Molecular neurobiology. vol 9. issue 1-3. 1995-04-18. PMID:7888094. |
neurocircuitry of the basal ganglia studied by monitoring neurotransmitter release. |
1995-04-18 |
2023-08-12 |
Not clear |
M Herrera-Marschitz, C F Loidl, Z B You, K Andersson, R Silveira, W T O'Connor, M Goin. Neurocircuitry of the basal ganglia studied by monitoring neurotransmitter release. Effects of intracerebral and perinatal asphyctic lesions. Molecular neurobiology. vol 9. issue 1-3. 1995-04-18. PMID:7888094. |
the neurocircuitries of the basal ganglia are studied with in vivo microdialysis, with special consideration to dopamine transmission and its interaction with other neurotransmitter systems. |
1995-04-18 |
2023-08-12 |
Not clear |
K W Lange, P Riederer, M B Youdi. Biochemical actions of l-deprenyl (selegiline). Clinical pharmacology and therapeutics. vol 56. issue 6 Pt 2. 1995-01-18. PMID:7995015. |
because parkinson's disease is characterized by a decrease in dopaminergic neurotransmission in the basal ganglia, the selective inhibition of mao-b should lead to diminished metabolism of dopamine in the nigrostriatal system and a significant increase in the concentration of the neurotransmitter. |
1995-01-18 |
2023-08-12 |
human |
C F Loidl, M Herrera-Marschitz, K Andersson, Z B You, M Goiny, W T O'Connor, R Silveira, R Rawal, B Bjelke, Y Che. Long-term effects of perinatal asphyxia on basal ganglia neurotransmitter systems studied with microdialysis in rat. Neuroscience letters. vol 175. issue 1-2. 1994-12-29. PMID:7970219. |
long-term effects of perinatal asphyxia on basal ganglia neurotransmitter systems studied with microdialysis in rat. |
1994-12-29 |
2023-08-12 |
rat |
A K Afif. Basal ganglia: functional anatomy and physiology. Part 1. Journal of child neurology. vol 9. issue 3. 1994-10-27. PMID:7930403. |
within the internal connectivity of the basal ganglia, there is a fast system in which the neurotransmitter is gamma-aminobutyric acid (gaba) and a slow system modulated by neuropeptides. |
1994-10-27 |
2023-08-12 |
Not clear |
D G Standaert, C M Testa, A B Young, J B Penne. Organization of N-methyl-D-aspartate glutamate receptor gene expression in the basal ganglia of the rat. The Journal of comparative neurology. vol 343. issue 1. 1994-08-10. PMID:8027428. |
glutamate is an important neurotransmitter in the circuitry of the basal ganglia. |
1994-08-10 |
2023-08-12 |
rat |
R K Schwarting, G Pei, S Söderström, T Ebendal, J P Husto. Unilateral stimulation or removal of rat vibrissae: analysis of nerve growth factor and tyrosine hydroxylase mRNA in the brain. Behavioural brain research. vol 60. issue 1. 1994-06-17. PMID:7910464. |
the results are discussed with respect to the interaction of vibrissae function with the basal ganglia, the neurotransmitter dopamine and mechanism of functional recovery. |
1994-06-17 |
2023-08-12 |
rat |
G L Wenk, M O'Leary, C B Nemeroff, G Bissette, H Moser, S Naid. Neurochemical alterations in Rett syndrome. Brain research. Developmental brain research. vol 74. issue 1. 1993-11-02. PMID:8403377. |
the results suggest that changes in specific neurotransmitter systems, particularly cholinergic neurons, in the thalamus, cerebellum and basal ganglia may underlie the progressive deterioration in motor and cognitive function characteristic of this disorder. |
1993-11-02 |
2023-08-12 |
Not clear |
S Suemaru, K Suemaru, K Kawai, Y Ihara, K Hashimot. [Cerebrospinal fluid and plasma corticotropin-releasing hormone (CRH) in dentatorubropallidoluysian atrophy (DRPLA)--study in two cases of hereditary DRPLA]. Rinsho shinkeigaku = Clinical neurology. vol 33. issue 4. 1993-10-14. PMID:8370212. |
together with the recent anatomic evidence on the distribution of crh neurons and receptors in the central nervous system, the present results suggest that degeneration of the subcortical structures including the basal nuclei, the brain stem and the cerebellum involves degeneration and/or dysfunction of the crh neuronal system which, presumably, are related to those of some other neurotransmitter systems, and that the reduced activity of the crh system plays a pathophysiological role in such diseases as drpla with degeneration of the subcortical nuclei and the cerebellum. |
1993-10-14 |
2023-08-12 |
Not clear |
B Bogert. Recent advances in the neuropathology of schizophrenia. Schizophrenia bulletin. vol 19. issue 2. 1993-08-03. PMID:8322039. |
but these studies also reported subtle changes in parts of the basal ganglia, thalamus, cortex, corpus callosum, and brainstem neurotransmitter systems. |
1993-08-03 |
2023-08-12 |
Not clear |
W Heindel, H Kugel, B Rot. Noninvasive detection of increased glycine content by proton MR spectroscopy in the brains of two infants with nonketotic hyperglycinemia. AJNR. American journal of neuroradiology. vol 14. issue 3. 1993-07-22. PMID:8517351. |
in patient 1 (49-day-old girl), the pathologic signal of the inhibitory neurotransmitter glycine was of similar size in the parietooccipital white matter and in the basal ganglia region. |
1993-07-22 |
2023-08-12 |
human |