| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| Z D Luo, S R Chaplan, B P Scott, D Cizkova, N A Calcutt, T L Yaks. Neuronal nitric oxide synthase mRNA upregulation in rat sensory neurons after spinal nerve ligation: lack of a role in allodynia development. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 19. issue 21. 1999-11-19. PMID:10531423. |
in rats with diabetic neuropathy, there was no significant change in drg nnos mrna. |
1999-11-19 |
2023-08-12 |
rat |
| M Hirade, H Yasuda, M Omatsu-Kanbe, R Kikkawa, H Kitasat. Tetrodotoxin-resistant sodium channels of dorsal root ganglion neurons are readily activated in diabetic rats. Neuroscience. vol 90. issue 3. 1999-06-14. PMID:10218793. |
to clarify the mechanism of hyperalgesia in diabetic neuropathy, we investigated the effects of streptozocin-induced hyperglycemia on tetrodotoxin-resistant na+ channel activity of dorsal root ganglion neurons. |
1999-06-14 |
2023-08-12 |
rat |
| J W Unger, T Klitzsch, S Pera, R Reite. Nerve growth factor (NGF) and diabetic neuropathy in the rat: morphological investigations of the sural nerve, dorsal root ganglion, and spinal cord. Experimental neurology. vol 153. issue 1. 1998-10-19. PMID:9743564. |
nerve growth factor (ngf) and diabetic neuropathy in the rat: morphological investigations of the sural nerve, dorsal root ganglion, and spinal cord. |
1998-10-19 |
2023-08-12 |
rat |
| J W Unger, T Klitzsch, S Pera, R Reite. Nerve growth factor (NGF) and diabetic neuropathy in the rat: morphological investigations of the sural nerve, dorsal root ganglion, and spinal cord. Experimental neurology. vol 153. issue 1. 1998-10-19. PMID:9743564. |
in order to further assess the effects of ngf on the somatosensory system in diabetic neuropathy, the sural nerve, dorsal root ganglia (drg), and dorsal horn of the spinal cord were investigated by morphological and quantitative methods in rats after 12 weeks of uncontrolled streptozotocin-induced diabetes mellitus. |
1998-10-19 |
2023-08-12 |
rat |
| T Sasaki, H Yasuda, K Maeda, R Kikkaw. Hyperalgesia and decreased neuronal nitric oxide synthase in diabetic rats. Neuroreport. vol 9. issue 2. 1998-04-29. PMID:9507963. |
to evaluate the role of nitric oxide synthase (nnos) in the pathogenesis of diabetic neuropathy, we investigated nociception and nnos expression in dorsal root ganglion (drg) of rats with streptozocin-induced diabetes. |
1998-04-29 |
2023-08-12 |
rat |
| P A Low, K K Nickander, H J Tritschle. The roles of oxidative stress and antioxidant treatment in experimental diabetic neuropathy. Diabetes. vol 46 Suppl 2. 1997-09-25. PMID:9285497. |
experimental diabetic neuropathy results in myelinopathy of dorsal roots and a vacuolar neuropathy of dorsal root ganglion. |
1997-09-25 |
2023-08-12 |
human |
| D W Zochodn. Neurotrophins and other growth factors in diabetic neuropathy. Seminars in neurology. vol 16. issue 2. 1997-04-11. PMID:8987129. |
diabetic neuropathy may be an ideal testing ground for these substances because specific neurotrophins and growth factors could theoretically prevent loss of diabetic dorsal root ganglion (drg) cells or enhance regeneration of diabetic nerves. |
1997-04-11 |
2023-08-12 |
human |
| K Sango, H Horie, A Okamura, S Inoue, T Takenak. Diabetes impairs DRG neuronal attachment to extracellular matrix proteins in vitro. Brain research bulletin. vol 37. issue 5. 1995-09-08. PMID:7633902. |
these results suggest that the cell-surface receptors, which enable drg neurons to bind to the extracellular matrix proteins, are impaired by diabetes, resulting in being one of the causes of diabetic neuropathy. |
1995-09-08 |
2023-08-12 |
mouse |
| D W Zochodne, L T Ho, J A Alliso. Dorsal root ganglia microenvironment of female BB Wistar diabetic rats with mild neuropathy. Journal of the neurological sciences. vol 127. issue 1. 1995-04-28. PMID:7699389. |
abnormalities in the microenvironment of dorsal root ganglia (drg) might play a role in the pathogenesis of sensory abnormalities in human diabetic neuropathy. |
1995-04-28 |
2023-08-12 |
human |