| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| H Mori, Y Motoi, T Kobayashi, M Hasegawa, A Yamamura, T Iwatsubo, Y Mizun. Tau accumulation in a patient with pallidonigroluysian atrophy. Neuroscience letters. vol 309. issue 2. 2001-10-11. PMID:11502352. |
we studied the brain of a patient with pallidonigroluysian atrophy (pnla) in whom argyrophilic and abnormally phosphorylated tau positive neurons and glia were identified in the brain on gallyas-braak silver staining and immunohistochemical analysis although neurofibrillary tangles were not seen by bodian silver stain. |
2001-10-11 |
2023-08-12 |
Not clear |
| K Miyamoto, A Ikemoto, I Akiguchi, R Kohno, T Imura, Y Fujit. A case of frontotemporal dementia and parkinsonism of early onset with progressive supranuclear palsy-like features. Clinical neuropathology. vol 20. issue 1. 2001-05-03. PMID:11220696. |
fibrillary changes were found in neurons and glia that were immunostained for tau. |
2001-05-03 |
2023-08-12 |
Not clear |
| T Uchihara, K Tsuchiya, A Nakamura, K Iked. Appearance of tau-2 immunoreactivity in glial cells in human brain with cerebral infarction. Neuroscience letters. vol 286. issue 2. 2000-07-14. PMID:10825646. |
this difference suggests that neurons and glia react differently to an ischemic insult by exhibiting different tau epitopes. |
2000-07-14 |
2023-08-12 |
human |
| S Yen, C Easson, P Nacharaju, M Hutton, S H Ye. FTDP-17 tau mutations decrease the susceptibility of tau to calpain I digestion. FEBS letters. vol 461. issue 1-2. 1999-12-14. PMID:10561502. |
frontal temporal dementia and parkinsonism linked to chromosome 17 (ftdp-17) is caused by splice site and missense mutations in the tau gene, and characterized by the accumulation of filamentous tau in cerebral neurons and glia. |
1999-12-14 |
2023-08-12 |
Not clear |
| D W Dickso. Neuropathologic differentiation of progressive supranuclear palsy and corticobasal degeneration. Journal of neurology. vol 246 Suppl 2. 1999-12-10. PMID:10525997. |
progressive supranuclear palsy (psp) and corticobasal degeneration (cbd) are usually sporadic multi-system degenerations associated with filamentous tau inclusions in neurons and glia. |
1999-12-10 |
2023-08-12 |
Not clear |
| K Noda, S Katayama, C Watanabe, Y Yamamura, S Nakamur. Gallyas- and tau-positive glial structures in motor neuron disease with dementia. Clinical neuropathology. vol 18. issue 5. 1999-11-19. PMID:10505430. |
since the regions where these structures appeared exhibited neuronal loss with gliosis, these data suggest that a cytoskeletal abnormality involving tau protein in glia might be associated with the degenerative process of mnd-d. |
1999-11-19 |
2023-08-12 |
Not clear |
| T Komori, N Shibata, M Kobayashi, S Sasaki, M Iwat. Inducible nitric oxide synthase (iNOS)-like immunoreactivity in argyrophilic, tau-positive astrocytes in progressive supranuclear palsy. Acta neuropathologica. vol 95. issue 4. 1998-06-12. PMID:9560010. |
although the precise relationship between no production and neuronal cell death in psp remained uncertain, based on the specificity of taf for psp brains, our results indicated a possible mechanism of no-mediated cytotoxicity that may contribute to the neuronal and glial cell damage followed by abnormal tau accumulation in this disease. |
1998-06-12 |
2023-08-12 |
Not clear |
| L Faff, A Reichenbach, J Albrech. Two modes of stimulation by ammonia of taurine release from cultured rabbit Müller cells. Neurochemistry international. vol 31. issue 2. 1997-09-02. PMID:9220463. |
a previous study revealed that a 10-min ('acute') treatment of cultured müller glia with ammonium ions (further referred to as 'ammonia') at 0.5-5 mm concentration stimulated the release of newly loaded taurine (tau) by a camp-dependent, osmoresistant mechanism. |
1997-09-02 |
2023-08-12 |
rabbit |
| L Faff, A Reichenbach, J Albrech. Two modes of stimulation by ammonia of taurine release from cultured rabbit Müller cells. Neurochemistry international. vol 31. issue 2. 1997-09-02. PMID:9220463. |
by contrast, a 10-min treatment with 65 mm kcl, which is a potent, camp-independent stimulus of tau release in untreated müller glia, produced no further enhancement of tau release in ammonia-, mb-, h7 or oca-10mg-pretreated cells. |
1997-09-02 |
2023-08-12 |
rabbit |
| L Faff, A Reichenbach, J Albrech. Ammonia-induced taurine release from cultured rabbit Müller cells is an osmoresistant process mediated by intracellular accumulation of cyclic AMP. Journal of neuroscience research. vol 46. issue 2. 1997-03-10. PMID:8915900. |
a previous study demonstrated the release of newly loaded radiolabelled taurine (tau) from cultured rabbit müller glia not only following typical cell volume-increasing treatments with high (65 mm) potassium ions or hypotonic media, but also with ammonium chloride (further referred to as ammonia), in a dose-dependent manner, at doses ranging from physiological (0.25 mm) to those accompanying hyperammonemic coma (5 mm) (faff-michalak et al., glia 10:114-120, 1994). |
1997-03-10 |
2023-08-12 |
rabbit |
| C Bergeron, M S Pollanen, L Weyer, S E Black, A E Lan. Unusual clinical presentations of cortical-basal ganglionic degeneration. Annals of neurology. vol 40. issue 6. 1997-02-13. PMID:9007094. |
cortical-basal ganglionic degeneration classically presents predominantly as a motor disorder with a unique constellation of histological alterations characterized by the presence of neuronal loss and gliosis in a selective distribution, swollen achromasic neurons, and extensive deposition of abnormal tau in neurons and glia. |
1997-02-13 |
2023-08-12 |
Not clear |
| T Hanihara, N Amano, T Takahashi, H Nagatomo, S Yagashit. Distribution of tangles and threads in the cerebral cortex in progressive supranuclear palsy. Neuropathology and applied neurobiology. vol 21. issue 4. 1996-01-11. PMID:7494600. |
in immunohistochemical studies, argentophilic glia and threads were mostly positive for tau 2, and a small number of them were weakly positive for ubiquitin and paired helical filament protein. |
1996-01-11 |
2023-08-12 |
Not clear |
| M Nishimura, H Tomimoto, T Suenaga, Y Namba, K Ikeda, I Akiguchi, J Kimur. Immunocytochemical characterization of glial fibrillary tangles in Alzheimer's disease brain. The American journal of pathology. vol 146. issue 5. 1995-06-15. PMID:7747799. |
the tangles in glial cells were immunolabeled by antibodies against tau and ubiquitin, and double immunocytochemistry analyzed by confocal laser scanning microscopy demonstrated that the cytoplasms of tangle-bearing glia were labeled by antibodies against transferrin and 2'3'-cyclic nucleotide 3'-phosphohydrolase. |
1995-06-15 |
2023-08-12 |
Not clear |
| U Lübke, J Six, M Villanova, J Boons, M Vandermeeren, C Ceuterick, P Cras, J J Marti. Microtubule-associated protein tau epitopes are present in fiber lesions in diverse muscle disorders. The American journal of pathology. vol 145. issue 1. 1994-08-08. PMID:7518193. |
although tau is predominantly a neuronal protein, it has been demonstrated in glia and other nonneuronal cells. |
1994-08-08 |
2023-08-12 |
Not clear |
| J Lowe, R J Mayer, M Lando. Ubiquitin in neurodegenerative diseases. Brain pathology (Zurich, Switzerland). vol 3. issue 1. 1994-01-28. PMID:8269084. |
neurofibrillary tangles in diverse diseases are associated with ubiquitin as are several other tau containing inclusions in both neurones and glia. |
1994-01-28 |
2023-08-12 |
Not clear |
| H Hayashi, Y Watanabe, T Shibuy. Relation between long-lasting amounts of excitatory amino acid and its neuronal uptake system in cultured cerebellar granule cells under hypoglycemia. Progress in neuro-psychopharmacology & biological psychiatry. vol 17. issue 3. 1993-05-17. PMID:8475325. |
moreover, the amounts of glu in a glia poor culture of granule cells tended to be higher than those in glia rich cultured cells, while gln, gly and tau concentrations were lower in glia poor cells than in glia rich cells. |
1993-05-17 |
2023-08-12 |
rat |
| H Hayashi, Y Watanabe, T Shibuy. Relation between long-lasting amounts of excitatory amino acid and its neuronal uptake system in cultured cerebellar granule cells under hypoglycemia. Progress in neuro-psychopharmacology & biological psychiatry. vol 17. issue 3. 1993-05-17. PMID:8475325. |
however, the amounts of glu at 10 and 14 div, and also gly and tau amounts at 10 div were significantly higher than those seen in the cultures of glia poor cells under normal conditions. |
1993-05-17 |
2023-08-12 |
rat |
| L I Binder, A Frankfurter, L I Rebhu. The distribution of tau in the mammalian central nervous system. The Journal of cell biology. vol 101. issue 4. 1985-11-07. PMID:3930508. |
tau localization was not observed in glia, which indicated that, at least in brain, tau is neuron specific. |
1985-11-07 |
2023-08-11 |
rat |