All Relations between microtubule-associated protein tau and neuroglial cell

Reference Sentence Publish Date Extraction Date Species
Radu Constantinescu, Henrik Zetterberg, Bj\\xc3\\xb6rn Holmberg, Lars Rosengre. Levels of brain related proteins in cerebrospinal fluid: an aid in the differential diagnosis of parkinsonian disorders. Parkinsonism & related disorders vol 15 issue 3 2009 18562238 the 42 amino acid fragment of amyloid-beta (abeta42), neurofilament light chain (nfl), neurofilament heavy chain (pnfh), tau protein, glial fibrillary acidic protein (gfap), neuron specific enolase (nse), s-100b protein, and myelin basic protein (mbp) are brain related proteins (brp) present in neurons and glia cells. 2009-04-20 2022-01-12 Not clear
Marisol Espinoza, Rohan de Silva, Dennis W Dickson, Peter Davie. Differential incorporation of tau isoforms in Alzheimer's disease. Journal of Alzheimer's disease : JAD vol 14 issue 1 2008 18525123 tau inclusions are found in neurons and occasionally glia in a variety of diseases. 2008-08-18 2022-01-12 Not clear
E Kuusisto, T Kauppinen, I Alafuzof. Use of p62/SQSTM1 antibodies for neuropathological diagnosis. Neuropathology and applied neurobiology vol 34 issue 2 2008 17961133 in the brain, pathological p62-positive aggregates comprise both cytoplasmic and nuclear types in neurones and glia, with abnormal tau, alpha-synuclein, tar dna-binding protein 43 or polyglutamine proteins as primary components. 2008-05-13 2022-01-12 Not clear
Dennis W Dickson, Rosa Rademakers, Michael L Hutto. Progressive supranuclear palsy: pathology and genetics. Brain pathology (Zurich, Switzerland) vol 17 issue 1 2007 17493041 characteristic tau pathology is also found in glia. 2007-06-26 2022-01-12 Not clear
Lisette Arnaud, Nikolaos K Robakis, Maria E Figueiredo-Pereir. It may take inflammation, phosphorylation and ubiquitination to 'tangle' in Alzheimer's disease. Neuro-degenerative diseases vol 3 issue 6 2007 16954650 in this review we focus on three topics relevant to ad pathology and to nft formation: (1) what triggers cns inflammation resulting in glia activation and neuronal toxicity; (2) how products of inflammation might change the substrate specificity of kinases/phosphatases leading to tau phosphorylation at pathological sites; (3) the relationship between the ubiquitin/proteasome pathway and tau ubiquitination and accumulation in nft. 2007-02-15 2022-01-12 Not clear
Rohan de Silva, Tammaryn Lashley, Catherine Strand, Anna-Maria Shiarli, Jing Shi, Jinzhou Tian, Kathryn L Bailey, Peter Davies, Eileen H Bigio, Kunimasa Arima, Eizo Iseki, Shigeo Murayama, Hans Kretzschmar, Manuela Neumann, Carol Lippa, Glenda Halliday, James MacKenzie, Rivka Ravid, Dennis Dickson, Zbigniew Wszolek, Takeshi Iwatsubo, Stuart M Pickering-Brown, Janice Holton, Andrew Lees, Tamas Revesz, David M A Man. An immunohistochemical study of cases of sporadic and inherited frontotemporal lobar degeneration using 3R- and 4R-specific tau monoclonal antibodies. Acta neuropathologica vol 111 issue 4 2006 16552612 patients with familial ftld associated with exon 10 n279k, n296h or +16 splice site mutations showed tau pathology characterised by neuronal neurofibrillary tangles (nft) and glial cell tangles that contained only 4r-tau isoforms, as did the nft in p301l mapt mutation. 2006-08-24 2022-01-12 Not clear
Chenghua Jin, Sadao Katayama, Masanori Hiji, Chigusa Watanabe, Koichi Noda, Shigenobu Nakamura, Masayasu Matsumot. Relationship between neuronal loss and tangle formation in neurons and oligodendroglia in progressive supranuclear palsy. Neuropathology : official journal of the Japanese Society of Neuropathology vol 26 issue 1 2006 16521479 progressive supranuclear palsy (psp) is a progressive degenerative disorder characterized by neuronal loss, gliosis and abnormal fibril formation of abnormally phosphorylated tau protein in neurons and glia cells, but the cause is not clear at present. 2006-04-07 2022-01-13 Not clear
Sara Johansson, Ann-Cathrin Rades\\xc3\\xa4ter, Richard F Cowburn, Johan Thyberg, Johan Luthma. Modelling of amyloid beta-peptide induced lesions using roller-drum incubation of hippocampal slice cultures from neonatal rats. Experimental brain research vol 168 issue 1-2 2006 16175362 the aim of this study was to establish an organotypic in vitro model for investigating effects of the amyloid beta (abeta)-peptide on pyramidal neuron degeneration, glial cell activation and tau phosphorylation. 2006-03-30 2022-01-13 Not clear
Deepa V Dabir, Michael B Robinson, Eric Swanson, Bin Zhang, John Q Trojanowski, Virginia M-Y Lee, Mark S Forma. Impaired glutamate transport in a mouse model of tau pathology in astrocytes. The Journal of neuroscience : the official journal of the Society for Neuroscience vol 26 issue 2 2006 16407562 filamentous tau inclusions in neurons and glia are neuropathological hallmarks of tauopathies. 2006-03-17 2022-01-12 Not clear
Mark S Forman, Devika Lal, Bin Zhang, Deepa V Dabir, Eric Swanson, Virginia M-Y Lee, John Q Trojanowsk. Transgenic mouse model of tau pathology in astrocytes leading to nervous system degeneration. The Journal of neuroscience : the official journal of the Society for Neuroscience vol 25 issue 14 2006 15814784 filamentous tau inclusions in neurons and glia are neuropathological hallmarks of sporadic and familial tauopathies. 2006-03-01 2022-01-12 Not clear
Yoshio Tsubo. [Clinical, genetic and pathological aspects of frontotemporal dementia and parkinsonism linked to chromosome 17 (FTDP-17)]. Rinsho shinkeigaku = Clinical neurology vol 44 issue 11 2005 15651319 microscopically, the neuropathologic hallmark is the presence of tau protein deposits in neurons or in both neurons and glia. 2005-03-04 2022-01-12 Not clear
Virginia M-Y Lee, Theresa K Kenyon, John Q Trojanowsk. Transgenic animal models of tauopathies. Biochimica et biophysica acta vol 1739 issue 2-3 2005 15615643 research advances in the last several decades have characterized and defined tau neuropathologies of both neuron and glia in these diverse disorders and this has stimulated development of animal models of tauopathies. 2005-02-03 2022-01-12 Not clear
Stuart Pickering-Brow. The tau gene locus and frontotemporal dementia. Dementia and geriatric cognitive disorders vol 17 issue 4 2004 15178931 a pathological hallmark in the brains from patients with tau mutations is an accumulation of insoluble tau within neurons and glia that is considered neurotoxic. 2004-09-24 2022-01-12 Not clear
K A Josephs, Y Tsuboi, D W Dickso. Creutzfeldt-Jakob disease presenting as progressive supranuclear palsy. European journal of neurology vol 11 issue 5 2004 15142229 the pathological substrate of psp consists of filamentous tau degenerative lesions affecting neurons and glia. 2004-06-28 2022-01-12 Not clear
S Taniguchi, A M McDonagh, S M Pickering-Brown, Y Umeda, T Iwatsubo, M Hasegawa, D M A Man. The neuropathology of frontotemporal lobar degeneration with respect to the cytological and biochemical characteristics of tau protein. Neuropathology and applied neurobiology vol 30 issue 1 2004 14720172 tau pathology, in the form of neurofibrillary tangles (nfts) and glial cell tangles, was present in six cases of ftd with parkinsonism linked to chromosome 17, five of these cases resulting from +16 splice-site mutation and one from +13 mutation in the tau gene. 2004-03-10 2022-01-13 Not clear
S Taniguchi, A M McDonagh, S M Pickering-Brown, Y Umeda, T Iwatsubo, M Hasegawa, D M A Man. The neuropathology of frontotemporal lobar degeneration with respect to the cytological and biochemical characteristics of tau protein. Neuropathology and applied neurobiology vol 30 issue 1 2004 14720172 one other case of ftd showed an unusual pathology characterized by massive extracellular deposition of tau protein, composed of 4-r tau isoforms, within white matter without neuronal or glial cell inclusions. 2004-03-10 2022-01-13 Not clear
Deepa V Dabir, John Q Trojanowski, Christiane Richter-Landsberg, Virginia M-Y Lee, Mark S Forma. Expression of the small heat-shock protein alphaB-crystallin in tauopathies with glial pathology. The American journal of pathology vol 164 issue 1 2004 14695329 thus, increased alphab-crystallin expression in glial tau inclusions may represent a response by glia to the accumulation of misfolded or aggregated tau protein that is linked to the pathogenesis of the glial pathology and distinct from mechanisms underlying neuronal tau pathology in neurodegenerative disease. 2004-02-27 2022-01-14 Not clear
Irene Litva. Update on epidemiological aspects of progressive supranuclear palsy. Movement disorders : official journal of the Movement Disorder Society vol 18 Suppl 6 issue 2004 14502655 psp is characterized by four-repeat tau aggregates in neurons (neurofibrillary tangles) and glia in specific basal ganglia and brainstem areas. 2004-02-24 2022-01-12 Not clear
Omi Katsuse, Eizo Iseki, Tetsuaki Arai, Haruhiko Akiyama, Takashi Togo, Hirotake Uchikado, Masanori Kato, Rohan de Silva, Andrew Lees, Kenji Kosak. 4-repeat tauopathy sharing pathological and biochemical features of corticobasal degeneration and progressive supranuclear palsy. Acta neuropathologica vol 106 issue 3 2004 12802605 although cbd and psp have a common pathological feature that 4r tau accumulates in neurons and glia, recent pathological studies have confirmed differences between the two disorders. 2004-01-09 2022-01-11 Not clear
Lawrence M Agiu. Are tau and alpha synuclein filamentous inclusions in neurons and glia analogous to accumulation of myosin/actin filaments in myofiber hypertrophy? Medical hypotheses vol 60 issue 3 2003 12581622 are tau and alpha synuclein filamentous inclusions in neurons and glia analogous to accumulation of myosin/actin filaments in myofiber hypertrophy? 2003-10-01 2022-01-11 Not clear
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