| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| Annarosa Arcangeli, Andrea Becchett. Complex functional interaction between integrin receptors and ion channels. Trends in cell biology. vol 16. issue 12. 2007-01-08. PMID:17064899. |
integrin receptors mediate adhesion of the cell to the extracellular matrix and thereby regulate cell motility, proliferation, differentiation and apoptosis. |
2007-01-08 |
2023-08-12 |
Not clear |
| Z Liu, H Li, X Wu, B H Yoo, S R Yan, A W Stadnyk, T Sasazuki, S Shirasawa, E C LaCasse, R G Korneluk, K V Rose. Detachment-induced upregulation of XIAP and cIAP2 delays anoikis of intestinal epithelial cells. Oncogene. vol 25. issue 59. 2007-01-04. PMID:16799641. |
detachment of normal epithelial cells from the extracellular matrix triggers apoptosis, a phenomenon called anoikis. |
2007-01-04 |
2023-08-12 |
Not clear |
| Hiroyuki Tsutsui, Tomomi Ide, Shintaro Kinugaw. Mitochondrial oxidative stress, DNA damage, and heart failure. Antioxidants & redox signaling. vol 8. issue 9-10. 2007-01-04. PMID:16987026. |
reactive oxygen species induce myocyte hypertrophy, apoptosis, and interstitial fibrosis by activating matrix metalloproteinases. |
2007-01-04 |
2023-08-12 |
Not clear |
| b' Ma\\xc5\\x82gorzata Krzystek-Korpacka, Ma\\xc5\\x82gorzata Matusiewicz, Teresa Bana\\xc5\\x9. [Structure and function of midkine, a novel heparin-binding growth factor]. Postepy higieny i medycyny doswiadczalnej (Online). vol 60. 2006-12-28. PMID:17115009.' |
the paper presents up-to-date views on the biological activity of midkine both at the cellular (mitogenic properties, participation in apoptosis, and cellular migration, adhesion, morphological differentiation, and chemokine synthesis stimulation) and tissue levels (participation in organogenesis, tissue regeneration and protection and in the formation and degradation of extracellular matrix). |
2006-12-28 |
2023-08-12 |
Not clear |
| André S Bachmann, James P Howard, Carl-Wilhelm Voge. Actin-binding protein filamin A is displayed on the surface of human neuroblastoma cells. Cancer science. vol 97. issue 12. 2006-12-19. PMID:16999820. |
our findings suggest new biological functions for filamins, including a role as mediators in anti-nb igm-induced apoptosis, and they add to the growing body of evidence of the interaction of cytoskeletal proteins with the extracellular matrix. |
2006-12-19 |
2023-08-12 |
human |
| M V Patrushev, V E Patrusheva, V A Kasymov, E V Evdokimovskiĭ, T E Ushakova, A I Gazie. [Release of mtDNA from mitochondria and activation of its replication in tissues of irradiated mice]. Tsitologiia. vol 48. issue 8. 2006-12-18. PMID:17147261. |
a sharp decrease in the amount of mtdna copies in the mitochondrial matrix of spleen cells on the first day after irradiation may be considered as apoptosis development. |
2006-12-18 |
2023-08-12 |
mouse |
| Rong-Ping Guo, Chong Zhong, Ming Shi, Chang-Qing Zhang, Wei Wei, Ya-Qi Zhang, Jin-Qing L. Clinical value of apoptosis and angiogenesis factors in estimating the prognosis of hepatocellular carcinoma. Journal of cancer research and clinical oncology. vol 132. issue 9. 2006-12-15. PMID:16763805. |
whereas some studies have indicated that the prognosis of hepatocellular carcinoma (hcc) was correlated to some apoptosis and angiogenesis factors: p53, survivin, matrix metalloproteinases (mmps, including mmp-2 and mmp-9) and vascular endothelial growth factor (vegf), other studies have failed to confirm this. |
2006-12-15 |
2023-08-12 |
Not clear |
| Zi-hui Wang, Ding Yu, Jie Zheng, Yan Che. [Proteome analysis of nuclear matrix proteins during arsenic trioxide induced apoptosis in K562 cells]. Beijing da xue xue bao. Yi xue ban = Journal of Peking University. Health sciences. vol 37. issue 2. 2006-12-12. PMID:15841146. |
[proteome analysis of nuclear matrix proteins during arsenic trioxide induced apoptosis in k562 cells]. |
2006-12-12 |
2023-08-12 |
Not clear |
| Richard Seonghun Nho, Hong Xia, Deanna Diebold, Judy Kahm, Jill Kleidon, Eric White, Craig A Henk. PTEN regulates fibroblast elimination during collagen matrix contraction. The Journal of biological chemistry. vol 281. issue 44. 2006-12-11. PMID:16963781. |
replicating physiologic apoptosis associated with tissue repair, fibroblasts incorporated into type i collagen matrices undergo apoptosis in response to collagen matrix contraction. |
2006-12-11 |
2023-08-12 |
Not clear |
| Richard Seonghun Nho, Hong Xia, Deanna Diebold, Judy Kahm, Jill Kleidon, Eric White, Craig A Henk. PTEN regulates fibroblast elimination during collagen matrix contraction. The Journal of biological chemistry. vol 281. issue 44. 2006-12-11. PMID:16963781. |
however, during subsequent collagen matrix contraction, the level of phosphorylated akt progressively declines, triggering apoptosis. |
2006-12-11 |
2023-08-12 |
Not clear |
| Richard Seonghun Nho, Hong Xia, Deanna Diebold, Judy Kahm, Jill Kleidon, Eric White, Craig A Henk. PTEN regulates fibroblast elimination during collagen matrix contraction. The Journal of biological chemistry. vol 281. issue 44. 2006-12-11. PMID:16963781. |
here we show that pten phosphatase becomes activated during collagen matrix contraction and is responsible for antagonizing phosphatidylinositol 3-kinase activity and promoting a decline in phosphorylated akt and fibroblast apoptosis in response to collagen contraction. |
2006-12-11 |
2023-08-12 |
Not clear |
| Richard Seonghun Nho, Hong Xia, Deanna Diebold, Judy Kahm, Jill Kleidon, Eric White, Craig A Henk. PTEN regulates fibroblast elimination during collagen matrix contraction. The Journal of biological chemistry. vol 281. issue 44. 2006-12-11. PMID:16963781. |
pten null fibroblasts displayed enhanced levels of phosphorylated akt and were resistant to collagen matrix contraction-induced apoptosis. |
2006-12-11 |
2023-08-12 |
Not clear |
| Patrick Laplante, Marc-André Raymond, Andrée Labelle, Jun-Ichi Abe, Renato V Iozzo, Marie-Josée Héber. Perlecan proteolysis induces an alpha2beta1 integrin- and Src family kinase-dependent anti-apoptotic pathway in fibroblasts in the absence of focal adhesion kinase activation. The Journal of biological chemistry. vol 281. issue 41. 2006-11-27. PMID:16882656. |
we have shown previously that apoptosis of ec triggers the proteolysis of extracellular matrix components and the release of a c-terminal fragment of perlecan, which in turn inhibits apoptosis of fibroblasts. |
2006-11-27 |
2023-08-12 |
Not clear |
| Patrick Laplante, Marc-André Raymond, Andrée Labelle, Jun-Ichi Abe, Renato V Iozzo, Marie-Josée Héber. Perlecan proteolysis induces an alpha2beta1 integrin- and Src family kinase-dependent anti-apoptotic pathway in fibroblasts in the absence of focal adhesion kinase activation. The Journal of biological chemistry. vol 281. issue 41. 2006-11-27. PMID:16882656. |
these results suggest that extracellular matrix fragments produced by apoptotic ec initiate a state of resistance to apoptosis in fibroblasts via an alpha2beta1 integrin/sfk (src and fyn)/phosphatidylinositol 3-kinase (pi3k)-dependent pathway. |
2006-11-27 |
2023-08-12 |
Not clear |
| Murray C H Clarke, Nichola Figg, Janet J Maguire, Anthony P Davenport, Martin Goddard, Trevor D Littlewood, Martin R Bennet. Apoptosis of vascular smooth muscle cells induces features of plaque vulnerability in atherosclerosis. Nature medicine. vol 12. issue 9. 2006-11-27. PMID:16892061. |
in contrast, vsmc apoptosis in atherosclerotic plaques of sm22alpha-hdtr apoe-/- mice induced marked thinning of fibrous cap, loss of collagen and matrix, accumulation of cell debris and intense intimal inflammation. |
2006-11-27 |
2023-08-12 |
mouse |
| Karim Si-Tayeb, Arnaud Monvoisin, Claire Mazzocco, Sébastien Lepreux, Marion Decossas, Gaëlle Cubel, Danièle Taras, Jean-Frédéric Blanc, Derrick R Robinson, Jean Rosenbau. Matrix metalloproteinase 3 is present in the cell nucleus and is involved in apoptosis. The American journal of pathology. vol 169. issue 4. 2006-11-24. PMID:17003494. |
matrix metalloproteinase 3 is present in the cell nucleus and is involved in apoptosis. |
2006-11-24 |
2023-08-12 |
human |
| Andrew C Newb. Do metalloproteinases destabilize vulnerable atherosclerotic plaques? Current opinion in lipidology. vol 17. issue 5. 2006-11-20. PMID:16960505. |
alternatively, metalloproteinases could destabilize vulnerable plaques by promoting matrix destruction, angiogenesis, leucocyte infiltration, and apoptosis. |
2006-11-20 |
2023-08-12 |
mouse |
| P S Hodkinson, T Elliott, W S Wong, R C Rintoul, A C Mackinnon, C Haslett, T Seth. ECM overrides DNA damage-induced cell cycle arrest and apoptosis in small-cell lung cancer cells through beta1 integrin-dependent activation of PI3-kinase. Cell death and differentiation. vol 13. issue 10. 2006-11-08. PMID:16410797. |
we demonstrate that extracellular matrix (ecm) activates phosphatidyl inositol 3-kinase (pi3-kinase) signaling in sclc cells and prevents etoposide-induced caspase-3 activation and subsequent apoptosis in a beta1 integrin/pi3-kinase-dependent manner. |
2006-11-08 |
2023-08-12 |
Not clear |
| R M Moore, J M Mansour, R W Redline, B M Mercer, J J Moor. The physiology of fetal membrane rupture: insight gained from the determination of physical properties. Placenta. vol 27. issue 11-12. 2006-11-03. PMID:16516962. |
work in the rat model has demonstrated that collagen remodeling, with activation of matrix metalloproteinases (mmps), and apoptosis increase markedly in the amnion at end-gestation, suggesting that these processes are involved in fetal membrane weakening. |
2006-11-03 |
2023-08-12 |
rat |
| S Bondini, Z M Younoss. Non-alcoholic fatty liver disease and hepatitis C infection. Minerva gastroenterologica e dietologica. vol 52. issue 2. 2006-10-26. PMID:16557185. |
these factors include mediators of metabolic syndrome such as adipokines, inflammatory cytokines, factors associated with oxidative stress, lipid peroxidation products, as well as apoptosis and hepatic stellate cell activation with the resultant deposition of extracellular matrix. |
2006-10-26 |
2023-08-12 |
Not clear |