| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| R Frlan, S Gobe. Inhibitors of cathepsin B. Current medicinal chemistry. vol 13. issue 19. 2006-09-11. PMID:16918357. |
it is involved in many physiological processes, such as remodeling of the extracellular matrix (wound healing), apoptosis, and activation of thyroxine and renin. |
2006-09-11 |
2023-08-12 |
Not clear |
| M Mukai, H Endo, T Iwasaki, M Tatsuta, A Togawa, H Nakamura, M Inou. RhoC is essential for TGF-beta1-induced invasive capacity of rat ascites hepatoma cells. Biochemical and biophysical research communications. vol 346. issue 1. 2006-08-28. PMID:16750170. |
transforming growth factor-beta1 (tgf-beta1) is a multifunctional growth factor that plays a role in cell proliferation, differentiation, extracellular matrix production, apoptosis, and cell motility. |
2006-08-28 |
2023-08-12 |
rat |
| Denise M T Yu, Xin M Wang, Geoffrey W McCaughan, Mark D Gorrel. Extraenzymatic functions of the dipeptidyl peptidase IV-related proteins DP8 and DP9 in cell adhesion, migration and apoptosis. The FEBS journal. vol 273. issue 11. 2006-08-23. PMID:16704418. |
we have previously shown that the in vitro roles of dipeptidyl peptidase iv and fibroblast activation protein in cell-extracellular matrix interactions and apoptosis are similarly independent of catalytic activity. |
2006-08-23 |
2023-08-12 |
Not clear |
| En-Hui Yao, Yi Yu, Noboru Fukud. Oxidative stress on progenitor and stem cells in cardiovascular diseases. Current pharmaceutical biotechnology. vol 7. issue 2. 2006-08-22. PMID:16724944. |
these effects include cell growth, apoptosis, migration, inflammatory gene expression and matrix regulation. |
2006-08-22 |
2023-08-12 |
Not clear |
| Susanne Rutschow, Jun Li, Heinz-Peter Schultheiss, Matthias Pauschinge. Myocardial proteases and matrix remodeling in inflammatory heart disease. Cardiovascular research. vol 69. issue 3. 2006-08-18. PMID:16417902. |
the myocardial inflammatory reaction not only affects myocardial hypertrophy and apoptosis, but it has a major influence on the regulation of extracellular matrix turnover. |
2006-08-18 |
2023-08-12 |
Not clear |
| Virna L Sales, George C Engelmayr, Bret A Mettler, John A Johnson, Michael S Sacks, John E Maye. Transforming growth factor-beta1 modulates extracellular matrix production, proliferation, and apoptosis of endothelial progenitor cells in tissue-engineering scaffolds. Circulation. vol 114. issue 1 Suppl. 2006-08-18. PMID:16820571. |
transforming growth factor-beta1 modulates extracellular matrix production, proliferation, and apoptosis of endothelial progenitor cells in tissue-engineering scaffolds. |
2006-08-18 |
2023-08-12 |
Not clear |
| Allen Cheng, Tom C Nguyen, Marcin Malinowski, Frank Langer, David Liang, George T Daughters, Neil B Ingels, D Craig Mille. Passive ventricular constraint prevents transmural shear strain progression in left ventricle remodeling. Circulation. vol 114. issue 1 Suppl. 2006-08-18. PMID:16820650. |
altered wall strain results in cytokine and reactive oxygen species production, which, in turn, stimulates apoptosis and extracellular matrix disruption and could be an important trigger for adverse global lv dilatation and remodeling. |
2006-08-18 |
2023-08-12 |
Not clear |
| Jaron Lockett, Shuping Yin, Xiaohua Li, Yonghong Meng, Shijie Shen. Tumor suppressive maspin and epithelial homeostasis. Journal of cellular biochemistry. vol 97. issue 4. 2006-08-14. PMID:16329135. |
it is further noted that extracellular maspin is sufficient to block tumor induced extracellular matrix degradation, tumor cell motility and invasion, whereas intracellular maspin is responsible for the increased cellular sensitivity to apoptosis. |
2006-08-14 |
2023-08-12 |
Not clear |
| Danyu Sun, Keith R McCra. Endothelial-cell apoptosis induced by cleaved high-molecular-weight kininogen (HKa) is matrix dependent and requires the generation of reactive oxygen species. Blood. vol 107. issue 12. 2006-08-14. PMID:16418331. |
endothelial-cell apoptosis induced by cleaved high-molecular-weight kininogen (hka) is matrix dependent and requires the generation of reactive oxygen species. |
2006-08-14 |
2023-08-12 |
Not clear |
| Danyu Sun, Keith R McCra. Endothelial-cell apoptosis induced by cleaved high-molecular-weight kininogen (HKa) is matrix dependent and requires the generation of reactive oxygen species. Blood. vol 107. issue 12. 2006-08-14. PMID:16418331. |
the ability of hka to induce endothelial-cell apoptosis is dependent on the generation of intracellular reactive oxygen species and associated with depletion of glutathione and peroxidation of endothelial-cell lipids, effects that occur only in cells cultured on matrix proteins permissive for hka-induced apoptosis. |
2006-08-14 |
2023-08-12 |
Not clear |
| Liezhen Fu, Akihiro Tomita, Hua Wang, Daniel R Buchholz, Yun-Bo Sh. Transcriptional regulation of the Xenopus laevis Stromelysin-3 gene by thyroid hormone is mediated by a DNA element in the first intron. The Journal of biological chemistry. vol 281. issue 25. 2006-08-11. PMID:16606608. |
the matrix metalloproteinase (mmp) stromelysin-3 (st3) (mmp11) was first isolated as a breast cancer-associated gene and is expressed in diverse human carcinomas and various developmental processes involving apoptosis. |
2006-08-11 |
2023-08-12 |
human |
| Min-Huey Chen, Jue-Long Wang, Chi-Yin Wong, Chung-Chen Yao, Yi-Jane Chen, Ching-Chuan Jian. Relationship of chondrocyte apoptosis to matrix degradation and swelling potential of osteoarthritic cartilage. Journal of the Formosan Medical Association = Taiwan yi zhi. vol 104. issue 4. 2006-08-01. PMID:15909064. |
relationship of chondrocyte apoptosis to matrix degradation and swelling potential of osteoarthritic cartilage. |
2006-08-01 |
2023-08-12 |
Not clear |
| Min-Huey Chen, Jue-Long Wang, Chi-Yin Wong, Chung-Chen Yao, Yi-Jane Chen, Ching-Chuan Jian. Relationship of chondrocyte apoptosis to matrix degradation and swelling potential of osteoarthritic cartilage. Journal of the Formosan Medical Association = Taiwan yi zhi. vol 104. issue 4. 2006-08-01. PMID:15909064. |
the purpose of this study was to investigate the correlation of chondrocyte apoptosis, matrix degradation, and the corruption of collagen architecture in the development of severe swelling of osteoarthritic cartilage. |
2006-08-01 |
2023-08-12 |
Not clear |
| Karin von Wnuck Lipinski, Petra Keul, Nicola Ferri, Susann Lucke, Gerd Heusch, Jens W Fischer, Bodo Levka. Integrin-mediated transcriptional activation of inhibitor of apoptosis proteins protects smooth muscle cells against apoptosis induced by degraded collagen. Circulation research. vol 98. issue 12. 2006-08-01. PMID:16728660. |
apoptosis of smooth muscle cells (smc) and degradation of the extracellular matrix (ecm) have both been implicated in atherosclerotic plaque rupture. |
2006-08-01 |
2023-08-12 |
Not clear |
| Wei-Shu Wang, Po-Min Chen, Huann-Sheng Wang, Wen-Yih Liang, Yeu S. Matrix metalloproteinase-7 increases resistance to Fas-mediated apoptosis and is a poor prognostic factor of patients with colorectal carcinoma. Carcinogenesis. vol 27. issue 5. 2006-07-31. PMID:16474169. |
matrix metalloproteinase-7 increases resistance to fas-mediated apoptosis and is a poor prognostic factor of patients with colorectal carcinoma. |
2006-07-31 |
2023-08-12 |
Not clear |
| L-J Guo, X-H Luo, H Xie, H-D Zhou, L-Q Yuan, M Wang, E-Y Lia. Tissue inhibitor of matrix metalloproteinase-1 suppresses apoptosis of mouse bone marrow stromal cell line MBA-1. Calcified tissue international. vol 78. issue 5. 2006-07-27. PMID:16691494. |
tissue inhibitor of matrix metalloproteinase-1 suppresses apoptosis of mouse bone marrow stromal cell line mba-1. |
2006-07-27 |
2023-08-12 |
mouse |
| Pierre H Vacho. [Cell survival: differences and differentiation]. Medecine sciences : M/S. vol 22. issue 4. 2006-07-25. PMID:16597413. |
the same complex nature applies with regards to anoikis, a form of apoptosis that is largely regulated by integrin-mediated, cell-extracellular matrix interactions. |
2006-07-25 |
2023-08-12 |
human |
| Allen Cheng, Frank Langer, Tom C Nguyen, Marcin Malinowski, Daniel B Ennis, George T Daughters, Neil B Ingels, D Craig Mille. Transmural left ventricular shear strain alterations adjacent to and remote from infarcted myocardium. The Journal of heart valve disease. vol 15. issue 2. 2006-07-25. PMID:16607903. |
alterations in transmural strains could be a trigger for this process, as these changes can produce apoptosis and extracellular matrix disruption. |
2006-07-25 |
2023-08-12 |
Not clear |
| C Burrows, J M P Holly, N J Laurence, E G Vernon, J V Carter, M A Clark, J McIntosh, C McCaig, Z E Winters, C M Perk. Insulin-like growth factor binding protein 3 has opposing actions on malignant and nonmalignant breast epithelial cells that are each reversible and dependent upon cholesterol-stabilized integrin receptor complexes. Endocrinology. vol 147. issue 7. 2006-07-24. PMID:16614079. |
consistent with this we have demonstrated that although igfbp-3 and a non-igf-binding fragment (serine phosphorylation domain peptide) reduced attachment and enhanced apoptosis of hs578t breast cancer cells cultured on collagen or laminin, it promoted their attachment and survival on fibronectin, which is abundant in the matrix of aggressive tumors. |
2006-07-24 |
2023-08-12 |
Not clear |
| Robert A Gatenby, Edward T Gawlinski, Arthur F Gmitro, Brant Kaylor, Robert J Gillie. Acid-mediated tumor invasion: a multidisciplinary study. Cancer research. vol 66. issue 10. 2006-07-18. PMID:16707446. |
this chronic exposure of peritumoral normal tissue to an acidic microenvironment produces toxicity by: (a) normal cell death caused by the collapse of the transmembrane h(+) gradient inducing necrosis or apoptosis and (b) extracellular matrix degradation through the release of cathepsin b and other proteolytic enzymes. |
2006-07-18 |
2023-08-12 |
Not clear |