| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| H I Roach, J Erenpreis. The phenotypic switch from chondrocytes to bone-forming cells involves asymmetric cell division and apoptosis. Connective tissue research. vol 35. issue 1-4. 1997-05-15. PMID:9084646. |
the crucial event in the phenotypic switch was an asymmetric cell division which resulted in one daughter cell which underwent apoptosis and another viable daughter cell which subsequently differentiated to an osteogenic cell, i.e to a smaller basophilic cell that was positive for alkaline phosphatase, type i collagen, osteonectin, osteopontin, bone sialoprotein and osteocalcin and that, after 12-15 days in culture, could synthesize a mineralized bone matrix within intact lacunae. |
1997-05-15 |
2023-08-12 |
chicken |
| G R Merto, N Cella, N E Hyne. Apoptosis is accompanied by changes in Bcl-2 and Bax expression, induced by loss of attachment, and inhibited by specific extracellular matrix proteins in mammary epithelial cells. Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research. vol 8. issue 2. 1997-05-06. PMID:9040947. |
apoptosis is accompanied by changes in bcl-2 and bax expression, induced by loss of attachment, and inhibited by specific extracellular matrix proteins in mammary epithelial cells. |
1997-05-06 |
2023-08-12 |
Not clear |
| M L Day, R G Foster, K C Day, X Zhao, P Humphrey, P Swanson, A A Postigo, S H Zhang, D C Dea. Cell anchorage regulates apoptosis through the retinoblastoma tumor suppressor/E2F pathway. The Journal of biological chemistry. vol 272. issue 13. 1997-05-02. PMID:9079623. |
we present evidence that in epithelial cells deprived of matrix contact, the growth suppression signal elicited by hypophosphorylated rb opposes stimulatory signals from serum growth factors, leading to a cell cycle conflict that triggers apoptosis. |
1997-05-02 |
2023-08-12 |
Not clear |
| T H Tezel, L V Del Prior. Reattachment to a substrate prevents apoptosis of human retinal pigment epithelium. Graefe's archive for clinical and experimental ophthalmology = Albrecht von Graefes Archiv fur klinische und experimentelle Ophthalmologie. vol 235. issue 1. 1997-04-16. PMID:9034841. |
harvesting human retinal pigment epithelium (rpe) for transplantation may separate the cells from their extracellular matrix and induce apoptosis. |
1997-04-16 |
2023-08-12 |
human |
| A López-Candales, D R Holmes, S Liao, M J Scott, S A Wickline, R W Thompso. Decreased vascular smooth muscle cell density in medial degeneration of human abdominal aortic aneurysms. The American journal of pathology. vol 150. issue 3. 1997-04-07. PMID:9060837. |
given the role that smcs normally play in maintaining medial architecture and in arterial wall matrix remodeling, the induction of smc apoptosis likely makes an important contribution to the evolution of aneurysm degeneration. |
1997-04-07 |
2023-08-12 |
human |
| M A Wrid. Cellular and molecular features of lens differentiation: a review of recent advances. Differentiation; research in biological diversity. vol 61. issue 2. 1997-04-01. PMID:8983174. |
areas of lens development reviewed here include: the induction and early embryology of the lens; lens cell culture techniques; the role of growth factors and cytokines; the involvement of gap junctions in lens cell-cell communication; the role of cell adhesion molecules, integrins, and the extracellular matrix; the role of the cytoskeleton; the processes of programmed cell death (apoptosis) and lens fibre cell denucleation; the involvement of pax and homeobox genes; and crystallin gene regulation. |
1997-04-01 |
2023-08-12 |
Not clear |
| K Aoshiba, S I Rennard, J R Spurze. Cell-matrix and cell-cell interactions modulate apoptosis of bronchial epithelial cells. The American journal of physiology. vol 272. issue 1 Pt 1. 1997-03-28. PMID:9038899. |
to determine whether cell-extracellular matrix (ecm) and cell-cell interactions modulate apoptosis in bronchial epithelium, we cultured human bronchial epithelial cells in different conditions and evaluated the cells for apoptosis. |
1997-03-28 |
2023-08-12 |
human |
| K Aoshiba, S I Rennard, J R Spurze. Cell-matrix and cell-cell interactions modulate apoptosis of bronchial epithelial cells. The American journal of physiology. vol 272. issue 1 Pt 1. 1997-03-28. PMID:9038899. |
plating cells on type i collagen, fibronectin, and biosynthesized matrix prevented apoptosis, due at least in part to integrin-mediated adhesion. |
1997-03-28 |
2023-08-12 |
human |
| D Aharoni, I Meiri, R Atzmon, I Vlodavsky, A Amsterda. Differential effect of components of the extracellular matrix on differentiation and apoptosis. Current biology : CB. vol 7. issue 1. 1997-03-25. PMID:8999995. |
differential effect of components of the extracellular matrix on differentiation and apoptosis. |
1997-03-25 |
2023-08-12 |
Not clear |
| B L Langill. Arterial remodeling: relation to hemodynamics. Canadian journal of physiology and pharmacology. vol 74. issue 7. 1997-03-19. PMID:8946070. |
the processes involved in arterial remodeling involve regulation of vascular cell migration and mitosis and apoptosis rates, control of matrix synthesis and degradation, and regulation of matrix reorganization. |
1997-03-19 |
2023-08-12 |
Not clear |
| M Zweyer, B M Riederer, R L Ochs, F O Fackelmayer, T Kohwi-Shigematsu, R Bareggi, P Narducci, A M Martell. Association of nuclear matrix proteins with granular and threaded nuclear bodies in cell lines undergoing apoptosis. Experimental cell research. vol 230. issue 2. 1997-03-14. PMID:9024791. |
association of nuclear matrix proteins with granular and threaded nuclear bodies in cell lines undergoing apoptosis. |
1997-03-14 |
2023-08-12 |
Not clear |
| S Pullan, J Wilson, A Metcalfe, G M Edwards, N Goberdhan, J Tilly, J A Hickman, C Dive, C H Streul. Requirement of basement membrane for the suppression of programmed cell death in mammary epithelium. Journal of cell science. vol 109 ( Pt 3). 1997-03-11. PMID:8907708. |
the correct type of extracellular matrix was required, since only a basement membrane, not plastic or a collagen i matrix, lowered the rate of apoptosis. |
1997-03-11 |
2023-08-12 |
mouse |
| A C Newby, S J Georg. Proliferation, migration, matrix turnover, and death of smooth muscle cells in native coronary and vein graft atherosclerosis. Current opinion in cardiology. vol 11. issue 6. 1997-03-10. PMID:8968671. |
it highlights the increasing importance attached to interactions between growth factors and components of the extracellular matrix in regulating migration and proliferation and the new roles assigned to apoptosis in these cells. |
1997-03-10 |
2023-08-12 |
Not clear |
| C H Lee, S K Lee, J G Chi, S C Park, S I Chung, M Saitoh, P Shrestha, M Mor. Immunohistochemical evaluation of transglutaminase C in tumours of salivary glands. European journal of cancer. Part B, Oral oncology. vol 32B. issue 6. 1997-03-07. PMID:9039224. |
transglutaminase c (tgase c), a family of ca(2+)-dependent enzymes and an essential component in the cross-linking of peptide bonds, has been found to be a marker of epithelial differentiation with a possible role in cellular apoptosis, extracellular matrix stabilisation and ca2+ binding, thereby having a potential role in tumour growth, differentiation and invasive behaviour. |
1997-03-07 |
2023-08-12 |
human |
| P Marchetti, D Decaudin, A Macho, N Zamzami, T Hirsch, S A Susin, G Kroeme. Redox regulation of apoptosis: impact of thiol oxidation status on mitochondrial function. European journal of immunology. vol 27. issue 1. 1997-03-05. PMID:9022031. |
our data indicate that thymocyte apoptosis induced by diamide mimics natural apoptosis in the sense that mitochondrial transmembrane potential (delta psi(m)) disruption precedes nuclear chromatin degradation; that monovalent thiol-reactive compounds inhibit apoptosis induced by diamide, glucocorticoids, irradiation, and topoisomerase inhibition; that the critical thiols determining cell fate after exposure to diamide, glucocorticoids, or dna damage are likely to be located in the mitochondrial matrix; and that thiol oxidation and reduction are critical for apoptosis induction by some stimuli (glucocorticoids, dna damage), but not by fas/cd95 cross-linking. |
1997-03-05 |
2023-08-12 |
Not clear |
| N Goberdhan, C Dive, C H Streul. The role of the extracellular matrix and lactogenic hormones in mammary gland apoptosis. Biochemical Society transactions. vol 24. issue 3. 1997-02-26. PMID:8878892. |
the role of the extracellular matrix and lactogenic hormones in mammary gland apoptosis. |
1997-02-26 |
2023-08-12 |
Not clear |
| G Scott, L Cassidy, A Busacc. Fibronectin suppresses apoptosis in normal human melanocytes through an integrin-dependent mechanism. The Journal of investigative dermatology. vol 108. issue 2. 1997-02-13. PMID:9008226. |
recent reports show that components of the extracellular matrix function as cell survival factors through the suppression of apoptosis (programmed cell death). |
1997-02-13 |
2023-08-12 |
human |
| G Scott, L Cassidy, A Busacc. Fibronectin suppresses apoptosis in normal human melanocytes through an integrin-dependent mechanism. The Journal of investigative dermatology. vol 108. issue 2. 1997-02-13. PMID:9008226. |
in this report we show that attachment to fibronectin suppresses apoptosis of normal human fetal and neonatal melanocytes in vitro and that prevention of attachment to underlying matrix or attachment to poly-l-lysine is a potent inducer of apoptosis in melanocytes. |
1997-02-13 |
2023-08-12 |
human |
| C C Solorzano, R Ksontini, J H Pruitt, P J Hess, P D Edwards, A Kaibara, A Abouhamze, T Auffenberg, R E Galardy, J N Vauthey, E M Copeland, C K Edwards, G Y Lauwers, M Clare-Salzler, S L MacKay, L L Moldawer, D D Lazaru. Involvement of 26-kDa cell-associated TNF-alpha in experimental hepatitis and exacerbation of liver injury with a matrix metalloproteinase inhibitor. Journal of immunology (Baltimore, Md. : 1950). vol 158. issue 1. 1997-01-30. PMID:8977217. |
our studies suggest that cell-associated tnf-alpha plays a role in the hepatocellular necrosis and apoptosis that accompany d-galn/lps- or con a-induced hepatitis, and that matrix metalloproteinase inhibitors are ineffective in preventing this hepatic injury. |
1997-01-30 |
2023-08-12 |
mouse |
| E E Furlong, N K Keon, F D Thornton, T Rein, F Marti. Expression of a 74-kDa nuclear factor 1 (NF1) protein is induced in mouse mammary gland involution. Involution-enhanced occupation of a twin NF1 binding element in the testosterone-repressed prostate message-2/clusterin promoter. The Journal of biological chemistry. vol 271. issue 47. 1997-01-13. PMID:8939902. |
overlaying the cells with laminin-rich extracellular matrix suppressed the apoptosis and the expression of the 74-kda nf1 and, in the presence of lactogenic hormones, initiated milk protein gene expression and the expression of two of the lactation-associated nf1 proteins (68 and 46 kda). |
1997-01-13 |
2023-08-12 |
mouse |