All Relations between microtubule-associated protein tau and neuroglial cell

Reference Sentence Publish Date Extraction Date Species
Mari Yoshid. [Neuropathology of tauopathy]. Brain and nerve = Shinkei kenkyu no shinpo vol 65 issue 12 2014 24323931 although psp and cbd sometimes share certain pathological distribution, which makes clinical diagnosis difficult, cellular tau pathology and aggregation patterns in neurons and glia are different between the two diseases. 2014-05-06 2022-01-12 Not clear
Naomi Kouri, Kenichi Oshima, Makio Takahashi, Melissa E Murray, Zeshan Ahmed, Joseph E Parisi, Shu-Hui C Yen, Dennis W Dickso. Corticobasal degeneration with olivopontocerebellar atrophy and TDP-43 pathology: an unusual clinicopathologic variant of CBD. Acta neuropathologica vol 125 issue 5 2013 23371366 corticobasal degeneration (cbd) is a disorder affecting cognition and movement due to a progressive neurodegeneration associated with distinctive neuropathologic features, including abnormal phosphorylated tau protein in neurons and glia in cortex, basal ganglia, diencephalon, and brainstem, as well as ballooned neurons and astrocytic plaques. 2013-10-31 2022-01-12 Not clear
Takahiko Tokud. [Biomarkers for the differential diagnosis of corticobasal syndrome]. Brain and nerve = Shinkei kenkyu no shinpo vol 65 issue 1 2013 23300103 abnormal accumulations of the microtubule-associated tau protein are found in both neurons and glia, and cbd is thus considered one of the tauopathies. 2013-09-03 2022-01-12 Not clear
Dennis W Dickso. Parkinson's disease and parkinsonism: neuropathology. Cold Spring Harbor perspectives in medicine vol 2 issue 8 2013 22908195 the tau pathology of psp also affects both neurons and glia. 2013-06-26 2022-01-12 Not clear
Agneta Mewes, Heike Franke, David Singe. Organotypic brain slice cultures of adult transgenic P301S mice--a model for tauopathy studies. PloS one vol 7 issue 9 2013 22984603 deposits of hyperphosphorylated tau, called neurofibrillary tangles (nfts), surrounded by activated glia are modeled in transgenic mice, e.g. 2013-03-05 2022-01-12 Not clear
Takaaki Mori, Jun Maeda, Hitoshi Shimada, Makoto Higuchi, Hitoshi Shinotoh, Shu-ichi Ueno, Tetsuya Suhar. Molecular imaging of dementia. Psychogeriatrics : the official journal of the Japanese Psychogeriatric Society vol 12 issue 2 2012 22712644 furthermore, beneficial outcomes of a\xce\xb2 and tau immunization in humans and mouse models have highlighted crucial roles of immunocompetent glia in the protection of neurons against amyloid toxicities. 2012-10-29 2022-01-12 Not clear
Yasumasa Kokubo, Akira Taniguchi, Masato Hasegawa, Yuma Hayakawa, Satoru Morimoto, Misao Yoneda, Yoshihumi Hirokawa, Taizo Shiraishi, Yuko Saito, Shigeo Murayama, Shigeki Kuzuhar. \\xce\\xb1-Synuclein pathology in the amyotrophic lateral sclerosis/parkinsonism dementia complex in the Kii Peninsula, Japan. Journal of neuropathology and experimental neurology vol 71 issue 7 2012 22710962 phosphorylated \xce\xb1-synuclein frequently colocalized with tau in neuron cell bodies, neurites, and glia. 2012-09-04 2022-01-12 Not clear
Yazi D Ke, Alexandra K Suchowerska, Julia van der Hoven, Dineeka M De Silva, Christopher W Wu, Janet van Eersel, Arne Ittner, Lars M Ittne. Lessons from tau-deficient mice. International journal of Alzheimer's disease vol 2012 issue 2012 22720190 both alzheimer's disease (ad) and frontotemporal dementia (ftd) are characterized by the deposition of hyperphosphorylated forms of the microtubule-associated protein tau in neurons and/or glia. 2012-08-23 2022-01-12 Not clear
Diana A T Nijholt, Elise S van Haastert, Annemieke J M Rozemuller, Wiep Scheper, Jeroen J M Hoozeman. The unfolded protein response is associated with early tau pathology in the hippocampus of tauopathies. The Journal of pathology vol 226 issue 5 2012 22102449 increased presence of upr activation markers pperk and pire1 was observed in neurons and glia in ftld-tau cases, in contrast to ftld subtypes negative for tau pathology or in non-neurological controls. 2012-04-27 2022-01-12 Not clear
Dennis W Dickson, Naomi Kouri, Melissa E Murray, Keith A Joseph. Neuropathology of frontotemporal lobar degeneration-tau (FTLD-tau). Journal of molecular neuroscience : MN vol 45 issue 3 2012 21720721 a clinically and pathologically heterogeneous type of frontotemporal lobar degeneration has abnormal tau pathology in neurons and glia (ftld-tau). 2012-03-19 2022-01-12 Not clear
Bradley F Boev. The multiple phenotypes of corticobasal syndrome and corticobasal degeneration: implications for further study. Journal of molecular neuroscience : MN vol 45 issue 3 2012 21853287 microscopically, abnormal accumulations of the microtubule-associated tau protein are found in both neurons and glia, and this disorder is now considered one of the "tauopathies." 2012-03-19 2022-01-12 Not clear
Arne Ittner, Yazi D Ke, Janet van Eersel, Amadeus Gladbach, J\\xc3\\xbcrgen G\\xc3\\xb6tz, Lars M Ittne. Brief update on different roles of tau in neurodegeneration. IUBMB life vol 63 issue 7 2012 21698753 both alzheimer's disease (ad) and almost every second case of frontotemporal lobar degeneration (ftld) are characterized by the deposition of hyperphosphorylated forms of the microtubule-associated protein tau in neurons and/or glia. 2012-01-11 2022-01-12 Not clear
Masaya Nagaishi, Hideaki Yokoo, Yoichi Nakazat. Tau-positive glial cytoplasmic granules in multiple system atrophy. Neuropathology : official journal of the Japanese Society of Neuropathology vol 31 issue 3 2011 21062361 these results indicate that tau-positive granules in glia are common findings in msa and that tau aggregation might be another pathway to neurodegeneration in msa. 2011-09-22 2022-01-12 Not clear
Saleta Sierra, Maria C Ramos, Pilar Molina, Cynthia Esteo, Jose Antonio V\\xc3\\xa1zquez, Javier S Burgo. Statins as neuroprotectants: a comparative in vitro study of lipophilicity, blood-brain-barrier penetration, lowering of brain cholesterol, and decrease of neuron cell death. Journal of Alzheimer's disease : JAD vol 23 issue 2 2011 21098985 we have made a comparative study of 9 statins (lovastatin, mevastatin, pravastatin, simvastatin, cerivastatin, atorvastatin, fluvastatin, pitavastatin, and rosuvastatin), analyzing several parameters that could be related to neuroprotection, such as chemical structure, lipophilicity, potential blood-brain-barrier penetration (bbb), 3-hydroxy-3-methylglutaryl co-enzyme a reductase inhibition, cholesterol modulation in neurons, glia, and human hepatocyte cell lines, and protection against neurodegeneration caused by tau hyperphosphorylation induced by okadaic acid. 2011-05-12 2022-01-12 Not clear
Kenneth J Colodner, Mel B Fean. Glial fibrillary tangles and JAK/STAT-mediated glial and neuronal cell death in a Drosophila model of glial tauopathy. The Journal of neuroscience : the official journal of the Society for Neuroscience vol 30 issue 48 2011 21123557 a subset of neurodegenerative tauopathies is characterized by abundant filamentous inclusions of hyperphosphorylated tau in both neurons and glia. 2011-01-06 2022-01-12 Not clear
Kenneth J Colodner, Mel B Fean. Glial fibrillary tangles and JAK/STAT-mediated glial and neuronal cell death in a Drosophila model of glial tauopathy. The Journal of neuroscience : the official journal of the Society for Neuroscience vol 30 issue 48 2011 21123557 in summary, we present a genetically tractable model of glial fibrillary tau tangle formation and identify jak/stat signaling as mediating the death of both glia and neurons in this model. 2011-01-06 2022-01-12 Not clear
Michael Riedel, Olaf Goldbaum, Christiane Richter-Landsber. alpha-Synuclein promotes the recruitment of tau to protein inclusions in oligodendroglial cells: effects of oxidative and proteolytic stress. Journal of molecular neuroscience : MN vol 39 issue 1-2 2010 19266322 oxidative stress followed by proteasomal inhibition caused the occurrence of larger thioflavin s-positive inclusions, immunoreactive for tau and alphab-crystallin, thus resembling glial cell inclusion bodies. 2010-02-12 2022-01-12 Not clear
Bess Frost, Rachel L Jacks, Marc I Diamon. Propagation of tau misfolding from the outside to the inside of a cell. The Journal of biological chemistry vol 284 issue 19 2009 19282288 tauopathies are neurodegenerative diseases characterized by aggregation of the microtubule-associated protein tau in neurons and glia. 2009-07-21 2022-01-12 Not clear
Bin Ji, Jun Maeda, Makoto Sawada, Maiko Ono, Takashi Okauchi, Motoki Inaji, Ming-Rong Zhang, Kazutoshi Suzuki, Kiyoshi Ando, Matthias Staufenbiel, John Q Trojanowski, Virginia M Y Lee, Makoto Higuchi, Tetsuya Suhar. Imaging of peripheral benzodiazepine receptor expression as biomarkers of detrimental versus beneficial glial responses in mouse models of Alzheimer's and other CNS pathologies. The Journal of neuroscience : the official journal of the Society for Neuroscience vol 28 issue 47 2009 19020019 moreover, levels of glial cell line-derived neurotrophic factor (gdnf) in astrocytes were correlated with astroglial pbr, except for increased gdnf in pbr(-) astrocytes in the model of ad-like tau pathology, thereby suggesting that pbr upregulation in astrocytes is an indicator of neurotrophic support. 2009-06-09 2022-01-12 Not clear
Yusuke Takatsuru, Masae Iino, Kohichi Tanaka, Seiji Ozaw. Contribution of glutamate transporter GLT-1 to removal of synaptically released glutamate at climbing fiber-Purkinje cell synapses. Neuroscience letters vol 420 issue 1 2009 17513050 the values of tau(w) in glt-1(-/-) mice and glast(-/-) mice were comparable to those estimated in wt mice in the presence of a potent blocker of glial glutamate transporters (2s,3s)-3-[3-(4-methoxybenzoylamino)benzyloxy]aspartate (pmb-tboa) at 10 and 100 nm, which reduced the amplitudes of glutamate transporter currents elicited by cf stimulation in bergmann glia to approximately 81 and approximately 28%, respectively. 2009-06-02 2022-01-12 Not clear
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